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Cervical Radiculopathy/Radiating Neck Pain El Paso, TX.

Cervical Radiculopathy/Radiating Neck Pain El Paso, TX.

Cervical radiculopathy happens when a pinched nerve in the neck (cervical spine) causes pain.

Radicular pain can extend beyond the neck and radiate down:

  • The shoulders
  • Arms
  • Fingers

This type of nerve compression also causes:

  • Weakness
  • Numbness
  • Tingling
  • Reflex problems

The neck consists of 8 pairs of nerves that control several motor (strength) and sensory (feel) functions.

The cervical nerve roots at the top send movement and feeling signals to the head and neck, and the nerves at the bottom enable motor and sensory function to the arms and hands.

 

11860 Vista Del Sol, Ste. 128 Cervical Radiculopathy/Radiating Neck Pain El Paso, TX.

 

If one or more of the spinal nerves in the neck gets pinched, it can disturb its ability to function correctly.

This results in radiating pain in the neck and other areas of the body.

This condition can affect anyone but usually affects middle-aged adults.

Men also tend to develop cervical radiculopathy more than women.

Causes

The natural aging process on the spine is what usually causes cervical radiculopathy.

The spine goes through the aging process just like the rest of the body and even more as it is the basis of our structure.

This process can lead to several degenerative spinal disorders, that include:

  • Cervical spondylosis (osteoarthritis)
  • Spinal stenosis
  • Herniated discs

When nerve passageways begin to narrow, intervertebral discs begin to protrude,� and bone spurs, caused by these disorders can put pressure on the nerves in the neck.

The condition can also be caused by a traumatic injury to the neck like whiplash or sports injury.

Rarely is it caused by an infection or spinal tumor.

 

11860 Vista Del Sol, Ste. 128 Cervical Radiculopathy/Radiating Neck Pain El Paso, TX.

 

Symptoms

The primary symptom is pain radiating from the neck down to the:

  • Shoulders
  • Arms
  • Hands
  • Fingers

The above is an example of sensory function, which is related to feeling.

In addition to sensory symptoms, radiculopathy can also cause motor dysfunction.

Motor dysfunction relates to muscles and movement.

Reflex changes in the neck and upper body and weakness are examples of motor dysfunction.

Diagnosis

A spine specialist/chiropractor has several tools to diagnose cervical radiculopathy.

First and foremost your medical history will be reviewed and then will be:

  • Asked to describe symptoms

A physical exam will be conducted�to recreate the pain in a controlled manner in the:

  • Neck
  • Shoulder
  • Arms

Example: Spurling�s maneuver, which gently rotates the head, while applying gentle pressure.

Once the information from the medical history and physical exam are done,�imaging tests such as an MRI�may be ordered so they can pinpoint the location of the nerve compression.

MRI scans show the soft tissues in the spine, including the nerves.

The doctor may request a pair of diagnostic tests called electromyogram (EMG) and nerve conduction exam if there are significant upper nerve arm and neck pain.

These tests help understand if there is nerve damage, the cause of the damage and if the symptoms are related to the nerve damage.

EMG and nerve conduction tests are usually performed together to help in the diagnosis.

 

Emergency Symptoms

Once the spine specialist confirms the diagnosis, they will develop a treatment plan to relieve the nerve compression or prevent it from getting worse.

Most cases are taken care of with non-surgical treatment, however, if the following occurs you should contact your doctor:

  • Neck pain does not improve with treatment in the time your doctor expects.
  • Pain worsens regardless of treatment

Or you develop new:

  • Numbness
  • Weakness in the
  • Neck
  • Arms
  • Upper body
  • Develop fever

If you experience symptoms in the lower body like:

  • Weakness in the leg
  • Difficulty walking
  • Lack of bowel/bladder function, then seek medical attention immediately.

These symptoms may indicate cervical myelopathy, a more severe condition.

Cervical myelopathy is the compression of the spinal cord.

When the spinal cord gets compressed, it can generate widespread spine issues and usually requires surgery.

 

Treatment Cervical Radiculopathy

Like most types of spine pain, a doctor will recommend trying one or more conservative treatments first.

Conservative treatments are nonsurgical means.

It�s important to understand that just because a treatment is considered conservative does not mean it is ineffective.

In fact, it�s quite the opposite. Most people with nerve compression in their neck respond well to conservative therapies.

Though research on the efficacy of conservative treatments for cervical radiculopathy has produced mixed results, findings show that these therapies help eliminate pain and other nerve-related symptoms (like numbness and muscle weakness) in 40-80% of people.

11860 Vista Del Sol, Ste. 128 Cervical Radiculopathy/Radiating Neck Pain El Paso, TX.

 

The following are the most common conservative treatments:
  • Over-the-counter medications, like acetaminophen (Tylenol) or nonsteroidal anti-inflammatory medications (ibuprofen, Motrin)
  • Prescription medications, like steroids (prednisone), neuropathic agents (gabapentin, pregabalin), and muscle relaxants (baclofen, cyclobenzaprine)
  • Wearing a neck brace
  • Physical therapy and exercise, to strengthen the neck muscles and improve motion
  • Cervical spinal traction, that can be performed during physical therapy
  • Avoiding strenuous activity, but don’t avoid all activity, as too much rest can exacerbate the injury and extend the recovery time

These conservative treatments can go on for 6 to 8 weeks. If there is no improvement or it gets worse, then a doctor may want to step you up to the next level.

This may include steroid injections.

Spinal Injections

Cervical epidural steroid injections are considered a second-line treatment for radiculopathy that is not responding to conservative therapy. These injections send a dose of anti-inflammatory medicine into a specific nerve root�s that can relieve pain.

The number of injections differs from patient to patient. A doctor will make recommendations based on the condition and response to the first injection.

If the first injection reduces the pain and symptoms, a second or third injection might not be necessary unless symptoms recur.

If more than one is needed, they are given 3 weeks between each injection.

Injections can help manage pain and inflammation, but cannot strengthen or improve the flexibility of the cervical muscles.

Because of this, a doctor may prescribe physical therapy, chiropractic or an exercise program to condition the neck muscles.

Surgery Considered

When surgery is needed it is considered a last resort option. This is not a guaranteed solution and there are risks and complications.

Different types of surgical approaches are available. These procedures can be performed minimally invasively in a hospital setting or an outpatient surgery center.

Discussing options with a doctor and whether you are a candidate for minimally invasive surgery or not, along with other types of surgery e.g. artificial disc, is a discussion that is different for everybody, as some patients have existing medical conditions that can increase risks and complications.

Anterior cervical discectomy and fusion (ACDF)

This approach is the most widely used surgical approach.

The surgeon makes an incision through the front of the neck and removes the damaged intervertebral disc, fills the empty space with spacers to restore the height and attaches spinal instrumentation (plate, screws) for stabilization.

A bone graft is then packed into and around the body spacers for bone ingrowth and healing.

Posterior cervical foraminotomy

Here, the surgeon accesses one or more levels of the cervical spine with an incision in the back of the neck.

Foraminotomy decompresses the nerve root by removing whatever is compressing the nerve like a bone or soft tissue.

The procedure opens/widens the neural foramen or the nerve passageway where the nerve exits the spinal canal.

Cervical artificial disc replacement (C-ADR)

Here an artificial disc device is implanted in the empty disc space.

C-ADR is like a shock absorber and enables healthy movement the way that an actual disc does.

 

Conclusion

A compressed nerve in your neck can lead to radiating pain. This pain can make it almost unbearable to do simple tasks, even moving the neck from side to side or just opening a jar. Conservative treatment like chiropractic and exercise can ease the pain of this condition and restore function. Fortunately, surgery is rarely necessary.


 

El Paso, TX Neck Pain Chiropractic Treatment

 

Alfonso J. Ramirez now retired, found follow-up treatment with Dr. Alex Jimenez for his neck pain. Mr. Ramirez experienced chronic pain and headaches, but after receiving chiropractic care, he found relief from his symptoms. Ever since that time, Alfonso Ramirez has continued to maintain the alignment of his backbone with Dr. Jimenez. Mr. Ramirez is grateful for the chiropractic care he’s received for his neck pain and for his knee and shoulder pain. Alfonso J. Ramirez recommends Dr. Alex Jimenez as the non-invasive pick for neck pain.


 

NCBI Resources

Approximately two-thirds of the population being affected by neck pain at any time throughout their lives. Pain that originates in the cervical spine, or upper spine, can be caused by numerous other spinal health issues. Joint disruption in the neck can generate a variety of other common symptoms, which include headaches, head pain, and migraines. Neck pain affects about 5 percent of the global population, according to statistics.

 

The Importance of Micronutrition in Vitamins El Paso, Texas

The Importance of Micronutrition in Vitamins El Paso, Texas

Mostly everyone takes their vitamins in any shape and form. It can be from the foods that we eat to the supplements and vitamins pills that we make throughout the day. We can get the boost of micronutrients in our bodies with vitamins and foods. It can help with our diets when we are trying to get a head start in our healthy lifestyle change through healthy, nutritious, whole foods. Without it, it can cause our bodies to react differently with ailments. For example, a person has a healthy lifestyle, but they are feeling out of place in their daily lives; it might be due to their low levels of the vitamins they are not taking. We will be taking a look at the micronutrition in vitamins and supplements in this two-part series for the body.

The Vitamins

Vitamins are essential for the body since we can�t produce them naturally. When we feel sluggish or horrible, it might be due to the low vitamin intake that we are missing to make us feel better. Here are some of the vitamins that can help your body if you feel a bit weak in life.

Vitamin A

nutritional-sources-of-vitamin-a

Vitamin A is a fat-soluble compound that is an essential nutrient for the body. It is stored in the liver for later use and is transferred to the tissues when needed. Vitamin A helps maintain the integrity and function of all surface tissues and the eyes. Vitamin A has two forms which are retinol and retinyl esters and provitamin A carotenoids. Retinol and retinyl esters health benefits can help prevent macular degeneration to your eyes, and with the provitamin A carotenoids can have potent antioxidants to fight off free radicals in your body.

Deficiencies: Having a Vitamin A deficiency can lead to blindness in the eyes of some people. Anyone who has a deficiency in Vitamin A can have skin issues like hyperkeratosis and acne. Not only that, but it can increase any infections as well as have pregnancy complications for pregnant women.

Food Sources: Here are some of the foods that are very rich with vitamin A that are all carotenoid-rich in both animals and plants.

  • Egg yolks
  • Beef liver
  • Cod liver oil
  • Salmon
  • Sweet potatoes
  • Carrots
  • Dandelion greens
  • Cabbage

Vitamin B

image.jpeg

All B vitamins are water-soluble, and your body can�t store them. They are used to reduce fatigue and boost your mood. There are 8 B vitamins are vital and have many essential functions for maintaining good health.

  • B1 (thiamine): Thiamine helps our bodies metabolism by helping convert nutrients into energy. Some food sources include pork, sunflower seeds, and wheat germ.
  • B2 (riboflavin): Riboflavin converts food into energy and acts as an antioxidant in the body. Some of the food sources that are high in riboflavin are organ meats, beef, and mushrooms.
  • B3 (niacin): Niacin plays a role in cellular signaling, metabolism and DNA productions, as well as repairs it as well. Some food sources include chicken, tuna, and lentils.
  • B5 (pantothenic acid): Like other B vitamins, pantothenic acid helps our bodies obtain energy from the food we eat. It also involves hormone and cholesterol production. Some food sources include liver, fish, yogurt, and avocado.
  • B6 (pyridoxine): Pyridoxine helps produces red blood cells in the body. It helps create amino acid metabolism and neurotransmitters for the body. Foods that are highly rich with this vitamin are chickpeas, salmon, and potatoes.
  • B7 (biotin): Biotin is highly essential for carbohydrate and fat metabolism and can regulate gene expression in the body. The best food sources that contain biotin are yeast, eggs, salmon, cheese, and liver.
  • B9 (folate): Our bodies need folate for cell growth, amino acid metabolism, the formation of red and white blood cells as well as proper cell division. Folate can be found in foods like leafy greens, liver, beans and in supplements like folic acid.
  • B12 (cobalamin): One of the best- known of all the B vitamins, B12 is vital for neurological function, DNA production, and red blood cell count. It can be found naturally in animal food sources like meats, eggs, seafood, and dairy.

Deficiencies: Even though taking the B vitamins are essential, there are side effects to taking a high dose of the vitamin, especially B3 and B6. Some of the side effects include vomiting, high blood sugar levels, skin lesions, nerve damage, and even liver damage.

Vitamin C

vitamin-c-1200x630.png

Vitamin C is one of the most essential vitamins since it can�t be produced by the body. It has so many roles and has been linked to many impressive health benefits. It can help boost antioxidant levels, reduce high blood pressure, and heart disease risk. It can protect your body against any gout attacks and reduce your risk of dementia while improving your iron absorption and boosting your immunity.

Deficiencies: When you don�t take enough vitamin C, it can raise up your blood sugar. Without it, you can develop scurvy if you don�t have enough of vitamin C in your system. You can get really sick, and your immune system will be shot if you don�t take vitamin C.

Food Sources: The most common way to get vitamin C in your body is through citrus fruit. There are lots of foods that contain vitamin C and are very delicious.

  • Red and green peppers
  • Oranges and orange juice
  • Kiwi
  • Guava
  • Broccoli
  • Strawberries
  • Brussel sprouts
  • Tomato juice
  • Cantaloupe

Vitamin D3

alternative-sources-of-vitamin-d

Also known as the sunshine vitamin, vitamin D3 is essential for maintaining healthy bones and teeth. It can support the immune system, brain, and nervous system by keeping it healthy. It will even regulate insulin levels and help managing diabetes. However, vitamin D3 can be synthesized to our body whenever sunlight hits our skin.

Deficiencies: Even though the body can create vitamin D, there many reasons that vitamin D deficiency can occur. If a person has a darker skin color and uses sunscreen to reduce the absorption of UVB (ultraviolet radiation B) rays from the sun. They can stop the production of vitamin D. Some of the symptoms of vitamin D deficiency include getting sick more, fatigue, muscle pain, and depression. And if it continues for long periods, it can lead to obesity, diabetes, hypertension, chronic fatigue syndrome, fibromyalgia, and osteoporosis, just to name a few.

Food Sources: There are a few foods that contain vitamin D naturally. And for vitamin D3 it is mostly animal produced.

  • Salmon
  • Sardines
  • Egg yolk
  • Shrimp
  • Milk (fortified)
  • Cereal (fortified)
  • Yogurt (fortified)

Vitamin E

Vitamin_E.jpg

Vitamin E is one of the most essential nutrients that is available as a dietary supplement and can occur naturally in foods. It is an antioxidant that can help protect your cells damage and is fat-soluble. Researchers have investigated that vitamin E can be used as a treatment for various degenerative diseases, including high blood pressure, heart disease, and cancer. It is a rare case for a vitamin E deficiency; however, it is a rare condition that is being researched.

Since Vitamin E is the most common nutrient found in most foods, here are some of the foods, including cooking oils that are exceptional.

  • Wheat Germ Oil
  • Sunflower seeds
  • Almonds
  • Hazelnut Oil
  • Goose meat
  • Peanuts
  • Mango

Conclusion

So with these vitamins, it can help your body feel so much better in the long run. Without them, our bodies will have various health problems that can hurt us in the long haul. When we take these vitamins, our bodies began to heal properly, and we can see that our moods are a bit better. We can have normal functions without the vitamins because we eat the food that contains them, but when we need that extra boost of energy, vitamins are the way to go.


Cites:

Basavaraj, K H, et al. �Diet in Dermatology: Present Perspectives.� Indian Journal of Dermatology, Medknow Publications, 2010, www.ncbi.nlm.nih.gov/pmc/articles/PMC2965901/.

Chiu, Zelia K, et al. �Patterns of Vitamin D Levels and Exposures in Active and Inactive Noninfectious Uveitis�Patients.� Ophthalmology, U.S. National Library of Medicine, 11 July 2019, www.ncbi.nlm.nih.gov/pubmed/31519386.

Choi, Hyon K, et al. �Vitamin C Intake and the Risk of Gout in Men: a Prospective Study.� Archives of Internal Medicine, U.S. National Library of Medicine, 9 Mar. 2009, www.ncbi.nlm.nih.gov/pubmed/19273781.

Ettarh, R R, et al. �Vitamin C Lowers Blood Pressure and Alters Vascular Responsiveness in Salt-Induced Hypertension.� Canadian Journal of Physiology and Pharmacology, U.S. National Library of Medicine, Dec. 2002, www.ncbi.nlm.nih.gov/pubmed/12564647.

Institute of Medicine (US) Panel on Micronutrients, Unknown. �Vitamin A.� Dietary Reference Intakes for Vitamin A, Vitamin K, Arsenic, Boron, Chromium, Copper, Iodine, Iron, Manganese, Molybdenum, Nickel, Silicon, Vanadium, and Zinc., U.S. National Library of Medicine, 1 Jan. 1970, www.ncbi.nlm.nih.gov/books/NBK222318/.

Kubala, Jillian. �Vitamin A: Benefits, Deficiency, Toxicity and More.� Healthline, 4 Oct. 2018, www.healthline.com/nutrition/vitamin-a.

Martel, Julianna L. �Vitamin B1 (Thiamine).� StatPearls [Internet]., U.S. National Library of Medicine, 14 Aug. 2019, www.ncbi.nlm.nih.gov/books/NBK482360/.

Megan Ware, RDN. �Vitamin D: Health Benefits, Facts, and Research.� Medical News Today, MediLexicon International, 13 Nov. 2017, www.medicalnewstoday.com/articles/161618.php.

Meyer-Ficca, Mirella, and James B Kirkland. �Niacin.� Advances in Nutrition (Bethesda, Md.), American Society for Nutrition, 16 May 2016, www.ncbi.nlm.nih.gov/pmc/articles/PMC4863271/.

N/A, Unknown. �Office of Dietary Supplements – Vitamin E.� NIH Office of Dietary Supplements, U.S. Department of Health and Human Services, 0AD, ods.od.nih.gov/factsheets/VitaminE-HealthProfessional/.

O’Leary, Fiona, and Samir Samman. �Vitamin B12 in Health and Disease.� Nutrients, Molecular Diversity Preservation International, Mar. 2010, www.ncbi.nlm.nih.gov/pmc/articles/PMC3257642/.

Ozuguz, Pinar, et al. �Evaluation of Serum Vitamins A and E and Zinc Levels According to the Severity of Acne Vulgaris.� Cutaneous and Ocular Toxicology, U.S. National Library of Medicine, June 2014, www.ncbi.nlm.nih.gov/pubmed/23826827.

Pham-Huy, Lien Ai, et al. �Free Radicals, Antioxidants in Disease and Health.� International Journal of Biomedical Science : IJBS, Master Publishing Group, June 2008, www.ncbi.nlm.nih.gov/pubmed/23675073.

Senoo, Haruki, et al. �Hepatic Stellate Cell (Vitamin A-Storing Cell) and Its Relative–Past, Present and Future.� Cell Biology International, U.S. National Library of Medicine, Dec. 2010, www.ncbi.nlm.nih.gov/pubmed/21067523.

Wong, Cathy. �Benefits of Vitamin C You May Not Know About.� Verywell Health, Verywell Health, 17 July 2019, www.verywellhealth.com/the-benefits-of-vitamin-c-supplements-89083.

Zempleni, Janos, et al. �Biotin.� BioFactors (Oxford, England), U.S. National Library of Medicine, 2009, www.ncbi.nlm.nih.gov/pubmed/19319844.

 

NMDA Excitotoxicity in Functional Neurology

NMDA Excitotoxicity in Functional Neurology

Excitotoxicity is a pathological mechanism seen in a variety of health issues where an excessive synaptic excitation causes neuronal death and is also believed to be caused by the extracellular accumulation of the excitatory neurotransmitter glutamate, which triggers and connects ionotropic N-methyl-D-aspartate glutamatergic receptors (NMDARs) in the brain. Generally, NMDARs regulate and maintain calcium in cells to help manage physiological mechanisms like synaptic plasticity and memory, however, excessive stimulation can ultimately increase intracellular calcium which triggers cell death signaling to activate apoptosis. This pathological mechanism has been suggested in a variety of health issues, such as traumatic brain injury (TBI) and Alzheimer’s disease (AD), where it is extensively examined to understand health issues and treatment approaches. In a stroke, excitotoxicity has been shown to be the main pathological mechanism where neuronal damage happens and it is considered to be a well-known goal for many recent attempts at developing stroke therapeutics. �

 

Stroke is an acute brain health issue which causes neuronal damage which has currently no safe and effective neuroprotective treatment approaches. Immediately following a stroke, the brain tissue loses blood perfusion and the center of the infarct deteriorates quickly. This then causes milder ischemia and many brain cells or neurons will result in delayed death which can take up to several hours or even days. Research studies show that the mechanism of cell death is mainly NMDA receptor-dependent excitotoxicity. In ischemic areas, extracellular glutamate levels increase while preventing glutamate release, synaptic activity, or NMDAR activation which was capable of limiting cell death in a variety of stroke models. Thus, preventing excitotoxicity is an important treatment approach for reducing brain damage and improving patient outcome measures following a stroke, and this has definitely encouraged extensive efforts towards developing NMDA receptor-based stroke treatment approaches over the last two decades. Unfortunately, these have largely met with rather disappointing results. Several research studies have failed to find the expected efficiency of NMDAR for decreasing brain injuries. The reasons behind the basic research study results and clinical trials are still unknown, however, several reasons have been suggested. These include, but are not limited to, the inability to utilize the correct doses necessary for neuroprotection due to their side-effects, the inability to use the drugs within their neuroprotective windows, poor experimental designs, and heterogeneity in the patient population. However, as we will briefly summarize in the following article, improvement in our understanding of the physiological and pathological mechanisms of NMDAR activation as well as the different pathways connected to different NMDAR subtypes, has allowed researchers to develop new treatment approaches which improve therapeutic windows and increase specificity for death signaling pathways, achieving neuroprotection without interrupting other essential signaling pathways downstream of the NMDAR receptor. �

 

Neuroprotectants Targeting NMDAR Subtypes

 

NMDAR subtypes have different purposes in excitotoxicity and physiology. The NMDAR is a receptor which generally has two GluN1, also known as NR1, subunits as well as two subunits from the GluN2 subfamily (GluN2A-2D, also known as NR2A-2D). In the cortex, the major subpopulations of NMDARs are GluN2A- or GluN2A and 2B-containing receptors. GluN2A-containing receptors are found in synapses whereas GluN2B-containing receptors are found on extrasynaptic membranes. GluN2A- and GluN2B-containing receptors are different from each other because they regulate and manage plasticity, favoring either long-term potentiation (GluN2A) or depression (GluN2B) through a variety of electrophysiological and pharmacological properties as well as signaling proteins. In addition, these receptors play a fundamental role in promoting cell survival (GluN2A) or death (GluN2B) after excitotoxic stimulation. Because GluN2A-containing receptors are mainly focused on synapses while GluN2B-containing receptors are focused to both synaptic and extrasynaptic membranes, when excitotoxic conditions cause glutamate to extend beyond synapses, GluN2B-mediated death signaling becomes stronger in comparison to survival signaling which ultimately results in death. Through a stroke, by way of instance, NMDARs are less likely to favor cell survival and can instead cause detrimental effects by preventing considerable normal physiological purposes. Selfotel, a non-specific NMDAR blocker, was neuroprotective against stroke in vitro and in vivo, however, it ultimately failed to be neuroprotective against stroke in clinical trials by causing a variety of intolerable side-effects. �

 

Treatment strategies to reduce undesirable side-effects, including glycine site antagonists and NMDAR subtype-specific improvements, was to target the allosteric glycine binding regions on the GluN1 subunits with licostinel and gavestinel instead of directly blocking the receptor. These drug candidates performed well in preclinical examinations, however, they also failed as a result of low efficiency despite minimal side-effect profiles. The negative side-effects were perhaps due to a missed window of time following a stroke that shows which receptor blockers are safe and effective in preventing death. �

 

Better treatment methods and techniques for reducing unwanted side-effects of NMDAR are to utilize the differences between their variations. By way of instance, the GluN2B-specific inhibitor traxoprodil is neuroprotective in stroke research studies and minimal side-effects, however, it has also failed in clinical trials. Similar to the glycine region antagonists, it possibly needs to be properly regulated and managed to function efficiently. GluN2A agonists should promote cell survival signaling which could allow recovery following a stroke as well as cell survival to prevent passing signaling. As a matter of fact, activation of GluN2A-containing receptors utilizing increased doses of glycine was neuroprotective in an animal model of stroke but further research studies must examine GluN2A activation as a treatment approach in human participants. �

 

While NMDAR antagonists and modulators are safe and effective at attenuating excitotoxicity in experimental versions, their shortcoming is the challenge in implementing treatment approaches early to coincide with the summit of excitotoxic glutamate release. Stroke patients frequently have no chance of receiving these treatment approaches in time. However, the health issue can be avoided if receptor blockers can be utilized in at-risk populations. One research study has shown that low doses of prophylactic memantine, an NMDAR non-competitive antagonist with few side-effects, can considerably decrease brain injury and functional deficits following a stroke. Whether any medications are tolerable, safe, and effective when taken this way remains to be demonstrated but innovative solutions may nevertheless address how to deliver those drugs. �

 

One factor apart from those of the failed clinical trials is the interplay of NMDARs in cell survival which may be completely misunderstood. In the last few decades, there has been accumulating evidence that synaptic NMDARs may also cause cell death and GluN2A, as well as GluN2B, do not necessarily have dichotomous functions in excitotoxicity. Further research studies may be required to demonstrate more nuanced receptor inhibitor strategies and to solve this controversy. �

 

Neuroprotectants Targeting Cell Death Signaling

 

A treatment approach for NMDAR inhibitors is to focus on the most downstream events for cell death which happen over a much longer time period following receptor activation. A variety of cell death pathways following activation have been determined and several groups have provided proof-of-principle evidence that these pathways can be regulated and managed with the utilization of peptides to ultimately protect brain cells or neurons without any side-effects. �

 

The oldest reported and most explored peptide strategy in stroke goals is nitrous oxide synthase (nNOS)-mediated cell death. NNOS connects to postsynaptic protein 95 (PSD95) which then connects to the C-terminal tail of the GluN2B subunit. NOS is a calcium-activated enzyme which activates the development of nitric oxide (NO) and its own status in the receptor complex which associates it in proximity to the focused stream of calcium entering activated GluN2B. In a stroke, the excessive calcium influx activates GluN2B-coupled nNOS. An interference peptide is utilized to disconnect the complex to prevent NO development. The peptide, Tat-NR2B9c, is made up of an HIV-1 Tat-derived cell penetration sequence which allows passage through the blood-brain barrier and cell membranes, connected to a copy of the region on the GluN2B for PSD95. The peptide and GluN2B disconnect PSD95, therefore, decoupling nNOS in the local considerable levels of calcium without interrupting the function of the receptor from different pathways. Utilization results in considerable protection against tissue and functional damage with no side-effects in vitro and in vivo after a single dose given before or after ischemia in vivo. The peptide has lately succeeded in Phase II clinical trial where it decreased iatrogenic infarcts during intracranial aneurysm treatment. This is the first time a research study has demonstrated efficiency in humans which also shows authenticity that targeting downstream cell death can be helpful against excitotoxic/ischemic neuronal injuries. �

 

While the utilization of peptides in a clinical setting is safe and effective, a similar efficiency has been achieved with small molecule drugs which act on the exact same goal and function like the peptides in a laboratory setting. To mimic Tat-NR2B9c, two small molecules, IC87201 and ZL006 have been individually demonstrated to compete at the identical GluN2B-specific connecting region without affecting the connection of PSD95 to other proteins. Additionally, ZL006 imitates the peptide’s neuroprotection without causing any considerable adverse side-effects. By identifying the goals and the specific regions, research studies can simulate small molecule drugs and accelerate their discovery towards excitotoxicity and stroke. �

 

Other GluN2B-specific pathways have been demonstrated in a similar manner and are showing promise in the stages of development. One such pathway which is triggered following GluN2B activation is the potentiation and recruiting of GluN2B in the cell membrane by death-associated protein kinase 1 (DAPK1). DAPK1 is a protein which connects to calmodulin to activate apoptosis but it is phosphorylated in an inactive form which is incapable of associating cell death and calmodulin. Following excitotoxicity, calcineurin activation dephosphorylates and triggers DAPK1, contributing to cell death. Furthermore, active DAPK1 can connect to and phosphorylate the C-terminal tail of receptors, excitotoxicity, and their function, aggravating calcium influx. A Tat-linked interference peptide which has the C-tail phosphorylation region which is GluN2B managed to block the interaction of active DAPK1 with GluN2B and promote excitotoxicity. Once the peptide was utilized in mice, dubbed Tat-NR2B-CT, it improved the outcome following ischemia. However, Tat-NR2B-CT was only efficient at preventing activity and runaway insertion instead of the downstream apoptotic of DAPK1 signaling. Researchers were also able to connect and guide DAPK1 towards lysosomes by including a sequence in the close of the hindrance peptide to create a degradation peptide. The result has been a serious and temporary fall in busy DAPK1 levels with a corresponding decrease in infarction when administering the peptide hours after ischemia, according to several research studies. �

 

The c-Jun N-terminal kinase 3 (JNK) acts upon many pathways and is a mediator for cell death in excitotoxicity. JNK interacting protein (JIP) connects and prevents JNK activity through a JNK binding domain (JBD) which spans over 20 residues. When these residues are connected to Tat as from the Tat-JBD20 interrupted peptide, they are capable of limiting JNK activity and preventing cell death in stroke models when administered before or after ischemia. The Tat-JBD20 peptide has also been shown utilizing D-amino acids instead of L-amino acids to withstand degradation by endogenous proteases. Doing so tremendously increases the peptide’s half-life and doesn’t negatively affect its binding affinity and selectivity, demonstrating that this alteration may be utilized for several interference peptides to boost efficiency and bioavailability. �

 

New targets are always being discovered. While currently, no new stroke treatment approaches are being utilized, a great deal of progress has been made by targeting the processes which occur during stroke towards creating treatment approaches. With the debut of the achievement of degradation and interruption peptides targeting GluN2B-specific passing signaling events, there’s hope that new treatments are on the horizon for health issues which have excitotoxicity. �

 

El Paso Chiropractor Dr. Alex Jimenez

Excitotoxicity is the pathological mechanism by which brain cells or neurons are ultimately damaged or eliminated by excessive stimulation from neurotransmitters, including glutamate and other similar substances. This ultimately occurs when the NMDA receptor and the AMPA receptor are overactivated by excitatory neurotransmitter glutamate receptors. This can cause a variety of processes which can damage cell structures, including components of the cytoskeleton, membrane, and DNA. Regulating and managing excitotoxicity can help maintain overall well-being. – Dr. Alex Jimenez D.C., C.C.S.T. Insight

 

Excitotoxicity is a pathological mechanism where an excessive synaptic excitation causes neuronal death and is also believed to be caused by the extracellular accumulation of the excitatory neurotransmitter glutamate, which triggers and connects ionotropic N-methyl-D-aspartate glutamatergic receptors (NMDARs) in the brain. This pathological mechanism has been suggested in a variety of health issues, such as traumatic brain injury (TBI) and Alzheimer’s disease (AD), where it is extensively examined to understand health issues and treatment approaches. The scope of our information is limited to chiropractic, musculoskeletal and nervous health issues as well as functional medicine articles, topics, and discussions. To further discuss the subject matter above, please feel free to ask Dr. Alex Jimenez or contact us at 915-850-0900 . �

 

Curated by Dr. Alex Jimenez �

 

References

 

  1. Li, Victor, and Yu Tian Wang. �Molecular Mechanisms of NMDA Receptor-Mediated Excitotoxicity: Implications for Neuroprotective Therapeutics for Stroke.� Neural Regeneration Research, Medknow Publications & Media Pvt Ltd, Nov. 2016, www.ncbi.nlm.nih.gov/pmc/articles/PMC5204222/.

 


 

Additional Topic Discussion: Chronic Pain

 

Sudden pain is a natural response of the nervous system which helps to demonstrate possible injury. By way of instance, pain signals travel from an injured region through the nerves and spinal cord to the brain. Pain is generally less severe as the injury heals, however, chronic pain is different than the average type of pain. With chronic pain, the human body will continue sending pain signals to the brain, regardless if the injury has healed. Chronic pain can last for several weeks to even several years. Chronic pain can tremendously affect a patient’s mobility and it can reduce flexibility, strength, and endurance.

 

 


 

Neural Zoomer Plus for Neurological Disease

Neural Zoomer Plus | El Paso, TX Chiropractor

Dr. Alex Jimenez utilizes a series of tests to help evaluate neurological diseases. The Neural ZoomerTM Plus is an array of neurological autoantibodies which offers specific antibody-to-antigen recognition. The Vibrant Neural ZoomerTM Plus is designed to assess an individual�s reactivity to 48 neurological antigens with connections to a variety of neurologically related diseases. The Vibrant Neural ZoomerTM Plus aims to reduce neurological conditions by empowering patients and physicians with a vital resource for early risk detection and an enhanced focus on personalized primary prevention. �

 

Formulas for Methylation Support

 

Xymogen Formulas - El Paso, TX

 

XYMOGEN�s Exclusive Professional Formulas are available through select licensed health care professionals. The internet sale and discounting of XYMOGEN formulas are strictly prohibited.

 

Proudly,�Dr. Alexander Jimenez makes XYMOGEN formulas available only to patients under our care.

 

Please call our office in order for us to assign a doctor consultation for immediate access.

 

If you are a patient of Injury Medical & Chiropractic�Clinic, you may inquire about XYMOGEN by calling 915-850-0900.

xymogen el paso, tx

 

For your convenience and review of the XYMOGEN products please review the following link.*XYMOGEN-Catalog-Download

 

* All of the above XYMOGEN policies remain strictly in force.

 


 

Triggers and Acute Lower Back Pain El Paso, Texas

Triggers and Acute Lower Back Pain El Paso, Texas

Sometimes low back pain comes out of nowhere, but that sudden twinge in the lower back does have a cause. With some cases, there�s a trigger, like picking up a heavy object/furniture from an awkward position.� But sometimes it can be a mystery and a challenge to diagnose.

It is important to know the cause of lower back pain to figure out the proper treatment plan. Otherwise, one could receive treatment for the wrong diagnosis and possibly exacerbate the existing injury.

Why do I need to know what triggers my back pain

Knowing what triggers back pain is the first step toward prevention and how to treat it.

Muscle spasms can stop you cold, and so I’m sure you don�t want another one.

 

11860 Vista Del Sol Ste. 128 Triggers and Acute Lower Back Pain El Paso, Texas

 

Acute Back Pain

Acute low back pain typically comes on suddenly and lasts for a short time.

It often resolves on its own with self-care and a little time.

Back pain that lasts longer than three months, is considered chronic.

Chronic back pain can be more complex and require doctor/spine specialist-directed treatment, like physical therapy.

Lower Back Pain Is Common

Over 90 percent of adults will have some type of low back pain during their life.

It happens to be the number one cause of job disability globally and the leading contributor to missed work.

Lower back pain happens more often, compared to mid or upper back pain because of the location and all of the movement.

The lower back supports the upper body’s weight.

The low back known as the (lumbar spine) absorbs and distributes all of the forces and stress when we move:

  • Walking
  • Standing
  • Rest
  • Sitting
  • Sleeping

Spinal and abdominal muscles that are weakened heighten injury risk.

These factors combined make the lower back vulnerable to painful spinal conditions.

Common Triggers

When the lumbar spine:

  • Muscles
  • Tendons
  • Ligaments

And other connective tissues get:

  • Pulled
  • Strained
  • Sprained

Is when lower back pain happens.

Small tears in the disc can also contribute to back pain.

Basically, any number of activity and non-activity can cause damage to the spinal discs depending on the movement.

A study published in Arthritis Care & Research saw 999 people from 300 clinics in Australia, to examine their pain triggers.

The most common triggers include:

Manual tasks performed in an awkward posture

This includes:

  • Lifting boxes with the back and not bending the knees
  • Lifting something too heavy
  • Moderate physical activity
  • Vigorous physical activity

High intense strength training, long bike rides without proper conditioning, handling people or animals, and picking up children can be triggers and cause injury.

Other triggers include:

  • Overstretching
  • Twisting
  • And Trauma

From:

  • Falls
  • Vehicle accidents
  • Sports

Triggers You Might Not Know About

Although we don’t think about it being Distracted can increase the odds of low back pain.

When we’re not paying attention,� we are more likely to lift and carry something too far from our body or distribute the weight unevenly.

Feeling tired and Fatigued is associated with lower back pain.

Getting the proper amount of sleep is vital to restore our bodies to optimal performance.

When we don’t sleep the right amount of hours we make our bodies more susceptible to injury.

Treatment

Thankfully most cases are not serious and typically resolve within a few days to, four weeks healing on its own.

But if not, then there are these conservative treatments that can help you feel better and speed up healing.

 

11860 Vista Del Sol Ste. 128 Triggers and Acute Lower Back Pain El Paso, Texas

 

Continue to Move Around

Depending on how much pain your body will allow, keep doing regular activities and exercise, as best as you can.

Activity increases blood flow, that moves oxygen and minerals/nutrients through the body.

Activity and movement help reduce muscle tension and inflammation.

Heat/Ice

This will not cure a strain or sprain, but they do help in pain reduction.

Heat helps loosen tight muscles.

This can be done by making warm compresses by soaking a towel in hot water.

Fold it to the size you need and wrap it around your lower back up to 20 minutes.� Then rest, massage and re-apply.

Therapy

Physical and manual therapies like:

If the pain is intense and interferes with daily activities, a chiropractor/physical therapist can offer exercises and stretches to:

  • Improve posture
  • Increase mobility
  • Correct muscle imbalances

Acute lower back pain can stop you cold with its intensity.

Understanding triggers can take steps to maintain a healthy spine and avoid unpleasant surprises.

Medication

Both acetaminophen (Tylenol) and non-steroidal anti-inflammatory drugs (NSAIDs) can relieve pain.

These need to be taken, specifically as directed by your doctor. We’ve seen the opioid crisis going on and now this type of treatment is now a last resort. Various medical associations are now pushing towards natural and alternative therapies before turning to medication.

Prolonged use of NSAIDs (Aleve, Advil) can be associated with an upset stomach, kidney damage and gastrointestinal conditions and bleeding, among other conditions.


 

Back Pain Specialist | El Paso, Tx

 

 

Back pain is one of the most common health issues frequently diagnosed by healthcare professionals. Approximately 80 percent of the population will experience some type of back pain throughout their lifetimes. Because back pain can occur due to a wide array of health issues, diagnosis is essential to follow-up with the proper treatment approach. Dr. Alex Jimenez, chiropractor or doctor of chiropractic in El Paso, TX, utilizes chiropractic care to help treat back pain. Patients describe how their back pain affected their quality of life, and how Dr. Jimenez helped them improve their overall health and wellness with chiropractic. Patients highly recommend Dr. Jimenez and his staff as the non-surgical choice for back pain, among other common health issues.


 

NCBI Resources

Throbbing, dull and achy, sharp and excruciating. All of these words can be used to describe lower back pain. Unfortunately, lower back pain is a common occurrence in adults. According to the�American Chiropractic Association, low back pain is the single leading cause of disability worldwide, with millions of reported cases every year. Patients who experience lower back pain never want to deal with it again, but�it can flare up periodically. According to the�National Institute of Neurological Disorders and Stroke,�roughly 20% of those who suffer from low back pain will eventually deal with it chronically. This can cause frustration, primarily when it affects mobility.

 

Intestinal Permeability and Functional Integrated Medicine Part: 2 El Paso, Texas

Intestinal Permeability and Functional Integrated Medicine Part: 2 El Paso, Texas

Today, we will be talking about what does the protein compounds and the peptide compounds do when a patient is being tested for food sensitivity. And we will also discuss what the Lectin and Dairy Zoomer do when a patient has a reaction to those types of food groups. In the last article, we mentioned about immunoglobulins in the intestinal barrier. And what do IgA and IgG antibodies do to the peptide and protein level?

Proteins vs. Peptides

So let us take a look at proteins and peptides since this is what Vibrant Food Zoomers are actually testing on a patient. Remember that the Food Zoomers are testing the peptides in the whole protein and testing all the links to see what the patient is actually sensitive to the foods they are consuming.

Proteins

Protein is basically abundant biomolecule that is consist of one or more long chains of amino acid residues. Proteins can be found in whole foods like meats and vegetables that can help the muscles in our bodies. In the last article, we talked about how IgA and IgG antibodies are used for food sensitivity testing.

iGen3_06-04_Figure-L

However, there is a limitation of a whole protein food sensitivity testing on a patient. Practitioners do make the assumption that the patient�s gut barrier is functional and intact since there are no signs of a leaky gut syndrome presented in the results. But, if that patient has the leaky gut syndrome, then the food sensitivity test will reflect what the patient has been eating. Another assumption is that the patient�s HCI and digestive enzymes are sufficient for tolerable proteolysis. Which means that those enzymes are breaking down whole proteins into smaller peptides.

Peptides

Peptides are what in protein molecules as they are short chains of amino acids and are linked by the peptide bonds. When they are being tested by the food sensitivity tests, the reproducibility is higher. It doesn�t rely on the excess HCI (hydrochloric acid) or enzymes. What the test eliminates is the cross-reactivity because peptides in proteins are not going to have molecular mimicry to other unrelated proteins.

The antibodies are highly specific to the peptides because they are not going to be generalized or more massive antibodies of proteins since cross-reactivity is eliminated. Another thing is that the peptide test does is that it can measure thousands of peptides in one protein for a full spectrum of reactivity.

Peptide-Bond-Formation

When patients are coming in with digestive problems and inflammatory condition/symptoms, practitioners take note that a lot of patients commonly have hypochlorhydria and deficiencies of enzymes and/or bile acids. Most patients sometimes have moderate to severe impairment of the intestinal barrier. When that happens, local doctors discuss with them that they may have to change their diets slowly but surely. And with integrative functional medicine that can occur.� Local practitioners look at their patient�s ailments and start detoxifying their bodies slowly. This helps their bodies heal and recommend them whole, nutritious, organic foods, and supplements to help repair the body naturally. Sometimes medicines can cause disruption to our bodies, however with whole natural foods and specific diets, it can help restore our bodies. Plus making sure that we exercise to make our bodies feel good and look good.

So now that we understand what proteins and peptides do when they are being tested. Let�s take a look at the food zoomers that can help you in case you have a sensitivity to these food groups. These are the Vibrant Lectin Zoomer and the Dairy Zoomer.

Lectin Zoomer

Screenshot 2019-09-23 11.05.59

The Lectin Zoomer is consist of a handful of lectins and a handful of aquaporins. The most common lectins that people consume are barley, bell pepper, chickpea, corn, cucumber, potato, etc. And the most common aquaporins that people consume are spinach, soybean, tomato, tobacco, etc.

Difference between Lectins and Aquaporins

The difference between lectins and aquaporins is that lectins are sugar-binding proteins that are found in both animals and plants, which can bind to the carbohydrate structures on cells. While aquaporins are water channels that are found in cavities in both plants and humans. Some aquaporins can cross-react and can lead to primarily neurological symptoms.

How Problematic are Lectins?

Some studies show cell toxicity in humans is done by using extreme cytotoxic lectins. Ricin, for example, is a common biological warfare element that is not from the commonly consumed legumes or grains. It contains cytotoxic lectins and is being consumed by animals like mice or pigs. The assumption is being made that there are similarities with humans and animal gut glycosylation (the process of sugar-binding) in these situations.

Unfortunately, though it hasn�t been demonstrated thoroughly. But lectins have biological activity in the human body. They have been used as a cancer treatment mechanism because they can agglutinate cancer cells. Which means that they produce cytotoxicity to cancer cells and can actually carry chemotherapy across cancer cell membranes.

Even though that is a good thing, lectins can facilitate the bacterial endotoxins across the epithelial barrier and go into the peripheral tissues. And that can cause inflammation to the intestinal epithelial barrier in the small intestines. Animals studies show that raw lectin consumptions can cause hemagglutinating effects, causing inflammation.

But we as humans don�t eat raw lectins because they are cooked, not pressurized cook. Certain foods that are lectins can be eaten raw or cooked. But animal studies stated that they are using for these studies are grain and legume lectins that are raw like beans and grains. But the upside is that lectins can affect the metabolism of nutrients to increase fat loss which is a positive side effect.

Measuring the Sensitivity to Lectins

On the Food Zoomers test, lectins are really not included in each analysis, except for the Wheat Zoomer. Surprisingly, a Food Zoomer may be non-reactive, but whoever is sensitive to a lectin component in the food they eat, may be reactive. So when that happens, it is necessary to eliminate the food temporarily.

If you are sensitive to a particular food, you can have a Food Zoomer and a lectin Zoomer combine. Because if you are sensitive to the food you consume, and it doesn�t show up on the Food Zoomer, but it shows up on the Lectin Zoomer. Then you should eliminate it from your diet for a bit until you retake the test.

Conditions Associated with Lectins

If you do have a lectin sensitivity, here are some of the terms that can affect your body.

  • Arthritis/rheumatoid arthritis
  • Connective tissue disorder
  • Gastrointestinal inflammation
  • Intestinal permeability
  • Possible cancer in established cancer patients

Now let�s take a look at the Dairy Zoomer and its functions if you are sensitive to whole dairy products.

Dairy Zoomer

The Dairy Zoomer is a peptide level assessment of the full spectrum of immune response possible to proteins in cow�s milk dairy. What this means that the Dairy Zoomer is only specific to cow�s milk. Since some proteins in cow�s milk are similar enough in the molecular structure to have the same homology to goat or sheep�s milk.

This means that these other kinds of milk may be potential can cause inflammatory in some individuals. The oral challenge for alternative types of fluid may be warranted, but use your best clinical judgment after the intestinal barrier is healed.

Screenshot 2019-09-23 11.00.24

What the Dairy Zoomer does is that it takes the milk protein and breaking each individual protein down to its different peptides. If you are wondering if the Dairy Zoomer is a test for lactose intolerance, it is not. Since lactose intolerance is not an immune-based reaction to dairy and does not involve any protein constituents of the food, therefore no antibodies are being generated.

What it is going to test for is the casein and whey proteins in the milk product from all animals, and the ratio of these proteins will vary by species. But all the proteins and milk will generally fall into one of these two proteins.

What to do with the results?

Doctor-Patient-Relationships-Unlocking-Doors-1

Once your patient comes back after taking the Food Zoomers test, here are some of the things to look for when you are retesting them.

  • If there are any IgA antibodies still in your patient, warrant an immediate elimination, regardless that it�s moderate or positive.
  • If there are any Moderate IgG antibodies in your patient, then it should be eliminated in the short term. Then rotate after a 30-60-day elimination and assessing the status of the intestinal permeability to confirm that that gut barrier is no longer �leaky.�
  • If there is a positive IgG result, then it should be eliminated long term and only reintroduced after 90+ days and confirm of an intact intestinal barrier.

Conclusion

So all in all, food sensitivity combine with the food zoomers test are an excellent way to help your body, especially the intestinal system. The Food Zoomers we used is functional for our patient�s wellness. Because we want to get rid of the excess antibodies and heal our patient�s body through the use of functional medicine.

 

Peripheral Neuropathy Relief & Treatment | El Paso, TX (2019)

Peripheral Neuropathy Relief & Treatment | El Paso, TX (2019)

Many healthcare professionals believe that peripheral neuropathy, which affects the peripheral nerves or the nerves which connect from the brain and spinal cord to the upper and lower extremities, can be permanent or irreversible. However, healthcare professionals like Dr. John Coppola and Dr. Valerie Monteiro have demonstrated that peripheral neuropathy can be treated through the utilization of a variety of treatment methods and techniques.

Dr. Coppola and Dr. Monteiro describe that because peripheral neuropathy can manifest due to a variety of health issues, such as diabetes, treating the underlying cause of a patient’s peripheral neuropathy can help treat their symptoms. The 5 critical keys for defeating peripheral neuropathy are ultimately described to help promote overall health and wellness. Dr. Alex Jimenez, a chiropractor in El Paso, Tx, can help ease symptoms associated with peripheral neuropathy. Dr. Alex Jimenez is the non-surgical choice for peripheral neuropathy.

 

Peripheral Neuropathy Relief & Treatment | El Paso, TX (2019)

 


Neuropathy is a medical term used to describe a collection of general diseases or malfunctions which affect the nerves. The causes of neuropathy, or nerve damage, can vary greatly among each individual and these may be caused by a number of different diseases, injuries, infections, and even vitamin deficiency states. However, neuropathy can most commonly affect the nerves that control the motor and sensory nerves. Because the human body is composed of many different kinds of nerves which perform different functions, nerve damage is classified into several types.

Neuropathy can also be classified according to the location of the nerves being affected and according to the disease-causing it. For instance, neuropathy caused by diabetes is called diabetic neuropathy. Furthermore, depending on which nerves are affected will depend on the symptoms that will manifest as a result. Below we will discuss several specific types of neuropathies clinically treated by chiropractors, physical therapists and physical medicine doctors alike, as well as briefly describing their causes and their symptoms.

Peripheral neuropathy, which is often simply referred to as �neuropathy,� is a state that happens when your nerves become damaged or injured, oftentimes simply disrupted. It�s estimated that neuropathy affects roughly 2.4 percent of the general populace and approximately 8 percent of people older than age 55. However, this quote doesn�t include people affected by neuropathy caused by physical trauma to the nerves.

 

Types

Neuropathy can affect any of the three types of peripheral nerves:

  • Sensory nerves, which transmit messages from the sensory organs, eyes, nose to the brain
  • Motor nerves, which track the conscious movement of the muscles
  • Autonomic nerves, which regulate the involuntary functions of the body

Sometimes, neuropathy will only impact one nerve. This is medically referred to as mononeuropathy and instances of it include:

  • Ulnar neuropathy, which affects the elbow
  • Radial neuropathy, which affects the arms
  • Peroneal neuropathy, which affects the knees
  • Femoral neuropathy, which affects the thighs
  • Cervical neuropathy, which affects the neck

Sometimes, two or more isolated nerves in separate regions of the body can become damaged, injured or disrupted, resulting in mono neuritis multiplex neuropathy. Most often, however, multiple peripheral nerves malfunction at the same time, a condition called polyneuropathy. According to the National Institute for Neurological Disorders and Stroke, or the NINDS, there are over 100 kinds of peripheral neuropathies.

 

Causes

Neuropathies are often inherited from birth or they develop later in life. The most frequent inherited neuropathy is the neurological disease Charcot-Marie-Tooth disease, which affects 1 in 2,500 people in the USA. Although healthcare professionals are sometimes not able to pinpoint the exact reason for an acquired neuropathy, medically referred to as idiopathic neuropathy, there are many known causes for them, including systemic diseases, physical trauma, infectious diseases, and autoimmune disorders.

A systemic disease is one which affects the whole body. The most frequent systemic cause behind peripheral neuropathy is diabetes, which can lead to chronically high blood glucose levels that harm nerves.

Other systemic issues can cause neuropathy, including:
  • Kidney disorders, which permit high levels of nerve-damaging toxic chemicals to flow in the blood
  • Toxins from exposure to heavy metals, including arsenic, lead, mercury, and thallium
  • Certain drugs and/or medications, including anti-cancer medications, anticonvulsants, antivirals, and antibiotics
  • Chemical imbalances because of liver ailments
  • Hormonal diseases, including hyperthyroidism, which disturbs metabolic processes, potentially inducing cells and body parts to exert pressure on the nerves
  • Deficiencies in vitamins, such as E, B1 (thiamine), B6 (pyridoxine), B12, and niacin, that can be vital for healthy nerves
  • Alcohol abuse, which induces vitamin deficiencies and might also directly harm nerves
  • Cancers and tumors that exert damaging pressure on nerve fibers and pathways
  • Chronic inflammation, which can damage protective tissues around nerves, which makes them more vulnerable to compression or vulnerable to getting inflamed and swollen
  • Blood diseases and blood vessel damage, which may damage or injure nerve tissue by decreasing the available oxygen supply

11860 Vista Del Sol Ste. 128 Peripheral Neuropathy Relief & Treatment | El Paso, TX (2019)

 

Signs and Symptoms

Depending on the reason and unique to each patient, signs, and symptoms of neuropathy can include:

  • Pain
  • Tingling
  • Burning/prickling sensations
  • Increased sensitivity to touch
  • Muscle weakness
  • Temporary or permanent numbness;
  • Paralysis
  • Dysfunction in glands or organs
  • Impairment in urination and
  • Sexual function

Such signs and symptoms are dependent on whether autonomic, sensory, or motor nerves, as well as a combination of them, are ultimately affected. Autonomic nerve damage can influence physiological functions like blood pressure or create gastrointestinal problems and issues. Damage or dysfunction in the sensory nerves may impact sensations and sense of equilibrium or balance, while harm to motor nerves may affect movement and reflexes. When both sensory and motor nerves are involved, the condition is known as sensorimotor polyneuropathy.

 

Complications

Peripheral�neuropathy�may result in several complications, as a result of disease or its symptoms. Numbness from the ailment can allow you to be less vulnerable to temperatures and pain, making you more likely to suffer from burns and serious wounds. The lack of sensations in the feet, for instance, can make you more prone to developing infections from minor traumatic accidents, particularly for diabetics, who heal more slowly than other people, including foot ulcers and gangrene.

Furthermore, muscle atrophy may cause you to develop particular physical disfigurements, such as pes cavus, a condition marked by an abnormally high foot arch, and claw-like deformities in the feet and palms.

 

Neuropathy Treatment

The first step in neuropathy treatment should be finding the root cause that’s causing the neuropathy.

Treatment of diseases such as:

  • Diabetes
  • Guillain-Barre syndrome
  • Rheumatoid arthritis
  • Sarcoidosis
  • Other underlying diseases

Prevents continued nerve damage and in some cases heals the damaged nerves.

If you are unaware of any underlying disease that is causing the peripheral neuropathy, make sure to let your doctor know of abnormal symptoms you may be experiencing.

Medication

Peripheral neuropathy can be treated with various medications.

The first type used to treat mild symptoms are:

  • Over-the-counter pain medications

In more severe cases:

  • Opiates
  • Narcotic medications
  • Anti-seizure medications

A doctor may prescribe a lidocaine patch or anti-depressants, as well to relieve symptoms.

Patients should thoroughly discuss medication for neuropathy treatment with a doctor before proceeding.

Physical Therapy

Physical therapy can benefit symptoms in neuropathy treatment.

A therapist will teach the patient exercises and stretches to help improve symptoms and increase muscle strength/control.

A therapist may also recommend braces or splints to improve mobility.

Patient’s should attend all physical therapy sessions to gain the maximum benefits.

 

Acids

Supplements like:

  • Essential acids called ALA (alpha-Lipoic acid)
  • GLA (gamma-linolenic acid) and omega-3 fatty acids

These can have a beneficial effect on diabetic peripheral neuropathy.

L-Carnitine

L-carnitine is a substance that the body makes and stores in the:

  • Liver
  • Brain

There have been reports that certain diabetics with neuropathy symptoms could regain regular sensation in the limbs when they increased their consumption of carnitine called acetyl-L-carnitine.

  • Red meat
  • Peanut butter
  • Dairy products

Are good dietary sources of this nutrient.

Supplements are also available at health food stores and pharmacies and health/wellness clinics.

 

Vitamins/Minerals

Vitamin deficiencies can result in peripheral neuropathy in some people.

Therefore there needs to be a replenishing of vitamins:

  • B
  • B12
  • E

Can help to decrease symptoms.

Recommended dosages are 300mg daily of vitamin E.

Doses of the different B vitamins differ, but one option for patients is to take a daily B-complex supplement.

 

Herbal Supplements

Herbal remedies are an alternative to explore.

St. John’s Wort, is a herbal supplement that can be taken orally and can reduce the pain.

Topical creams that have capsaicin, which is an anti-inflammatory found in chili peppers, can reduce the burning sensation.

 

Traditional Chinese Medicine TCM

Acupuncture can be an effective way to manage peripheral neuropathy.

Acupuncture uses pressure points throughout the body to realign the body’s energy, called the qi or chi.

Also, movement therapy is a way to manage the condition.

Tai chi and yoga can also help:

  • Align the body
  • Mind
  • Encourage relaxation
  • Distract from the pain

Even if the neuropathy treatment is only temporary, it can still help.


We are blessed to present to you�El Paso�s Premier Wellness & Injury Care Clinic.

Our services are specialized and focused on injuries and the complete recovery process.�Our areas of practice includeWellness & Nutrition, Chronic Pain,�Personal Injury,�Auto Accident Care, Work Injuries, Back Injury, Low�Back Pain, Neck Pain,�Migraine�Treatment, Sports Injuries,�Severe Sciatica, Scoliosis, Complex Herniated Discs,�Fibromyalgia, Chronic Pain, Stress Management, and Complex Injuries.

As El Paso�s Chiropractic Rehabilitation Clinic & Integrated Medicine Center,�we passionately are focused on treating patients after frustrating injuries and chronic pain syndromes. We focus on improving your ability through flexibility, mobility and agility programs tailored for all age groups and disabilities.

We want you to live a life filled with more energy, positive attitude, better sleep, less pain, proper body weight and educated on how to maintain this way of life.


Let Us Help You Get Back To Your Normal Life!

11860 Vista Del Sol Ste. 128 Peripheral Neuropathy Relief & Treatment | El Paso, TX (2019)


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NCBI Resources

Neuropathy can be caused by a variety of injuries and/or aggravated conditions, often manifesting into a plethora of associated signs and symptoms. While every type of neuropathy, such as diabetic neuropathy or autoimmune disease-associated neuropathy, develops its own unique group of signs and symptoms, many patients will often report common complaints. Individuals with neuropathy generally describe their pain as stabbing, burning or tingling in character.

If you experience unusual or abnormal tingling or burning sensations, weakness and/or pain in your hands and feet, it�s essential to seek immediate medical attention in order to receive a proper diagnosis of the cause of your specific signs and symptoms. Early diagnosis may help prevent further nerve injury. Visit http://www.neuropathycure.org�for more details.

 

 

Integrative Neurology: Traumatic Brain Injury and Alzheimer’s Disease

Integrative Neurology: Traumatic Brain Injury and Alzheimer’s Disease

The most common causes of TBI which result in ER visits include slip-and-fall accidents, blows to the head, and automobile accidents. Abrupt forces which jolt the brain violently within the skull, such as shock waves from explosions, which can also cause TBI. Traumatic brain injury can also result from bullet wounds or other injuries which penetrate the skull and brain. �

 

Doctors characterize traumatic brain injury as mild, moderate, or severe depending on whether the injury causes unconsciousness, how long it lasts, and other symptoms. Although most traumatic brain injuries are characterized as mild because they’re not considered life-threatening, even a mild TBI can have serious and long-lasting effects if left untreated. � Resulting from an impact to the head which interrupts brain function, TBI is a threat to cognitive health in two ways: �

 

  • The effects of traumatic brain injury, which may be long-lasting or even permanent, can include unconsciousness, inability to recall the event, confusion, difficulty learning new information, trouble speaking, unsteadiness, lack of coordination, and health issues associated with vision or hearing, among other common symptoms.
  • TBI may increase the risk of developing Alzheimer’s disease or dementia, years after the injury takes place.

 

According to the Centers for Disease Control and Prevention (CDC), approximately 2.8 million TBI-associated ER visits, hospitalizations, and deaths occurred in 2013, the latest year for which information is available. The purpose of the following article is to discuss traumatic brain injury (TBI) and its connection with Alzheimer’s disease and other health issues. �

 

Traumatic Brain Injury Causes

 

Slip-and-fall accidents are the most common cause of traumatic brain injury, where falls pose a potentially serious risk factor for older adults. According to a CDC special report evaluating data from several federal agencies, approximately 56,000 seniors are hospitalized every year as a result of head injuries sustained in falls. A serious TBI from a slip-and-fall accident may ultimately result in long-term cognitive changes and reduced ability to function as well as overall mood changes. �

 

About 775,000 older adults have traumatic brain injury-related disability. Measures to reduce the risk of falls include: �

 

  • Using a walker or other assistive device to compensate for mobility problems, muscle weakness or poor balance.
  • Having your vision checked regularly and using glasses or contact lenses that correct for changes.
  • Working with your doctor to watch for medication side effects or interactions among drugs you�re taking.
  • Avoiding household hazards, such as clutter, loose rugs or poor lighting.

 

Automobile accidents are another common cause of traumatic brain injury (TBI). People can reduce the risk of being involved in an auto accident by keeping their vehicle in good condition, following the rules of the road, and buckling their seat belt. Wearing a helmet and when biking, inline skating, or playing contact sports can also help protect the head from TBI. �

 

TBI Symptoms

 

The severity of symptoms for traumatic brain injuries largely depends on whether the injury is mild, moderate, or severe. Mild traumatic brain injury (TBI), also known as a concussion, can either not cause unconsciousness or can cause unconsciousness which lasts for 30 minutes or less. Mild traumatic brain injury (TBI) symptoms may include: �

 

  • Inability to remember the traumatic event immediately before or up to 24 hours after
  • Confusion and disorientation
  • Difficulty learning new information
  • Headache
  • Dizziness
  • Blurry vision
  • Nausea and vomiting
  • Ringing in the ears
  • Trouble speaking coherently
  • Mood changes or changes in sleeping patterns

 

These symptoms will commonly manifest at the time of the TBI or soon after, however, these may sometimes not develop till several days or even weeks following the traumatic event. Mild TBI symptoms are generally temporary and these will clear up within hours, days, or weeks following the traumatic even, however, they can occasionally last several months or longer. �

 

Moderate traumatic brain injury can cause unconsciousness which lasts more than 30 minutes but less than 24 hours and severe traumatic brain injury can cause unconsciousness for more than 24 hours. Symptoms of moderate and severe traumatic brain injury are similar to those of mild traumatic brain injury but these are more serious and longer-lasting. �

 

In all types of TBI, cognitive changes are the most common symptoms. The ability to learn and remember new information is also frequently affected. Other commonly affected cognitive skills include the ability to pay attention, organize thoughts, plan effective strategies for completing tasks and activities, and/or make sound judgments. More severe changes in cognitive skills may develop years after the traumatic event where the person may appear to have recovered from the previous TBI. �

 

TBI Diagnosis

 

Evaluations performed by healthcare professionals to help diagnose traumatic brain injury (TBI) generally include: �

 

  • Questions about the traumatic event
  • Analysis of the person’s level of consciousness and confusion
  • Neurological tests to analyze memory and thinking, vision, hearing, touch, balance, and reflexes

 

Let your doctor know if you are taking any drugs and/or medications, especially blood thinners, because they can increase the chance of complications. Also, inform your healthcare professional if you drink alcohol or take illicit drugs. �

 

Depending on the cause of the TBI and the severity of symptoms, brain imaging with computed tomography (CT) may be necessary to determine if there�s swelling or bleeding in the brain. If you experience a traumatic brain injury, it should be noted in your permanent medical record and mentioned whenever familiarizing a new doctor with your medical history. �

 

Traumatic Brain Injury Treatment

 

The most serious traumatic brain injuries commonly require specialized hospital care and can also need several months of rehabilitation. Most traumatic brain injuries are mild and can be treated with either a short hospital stay for observation or at-home monitoring followed by outpatient rehabilitation, if necessary. Treatment of dementia in a person with a history of traumatic brain injuries varies depending on the type of dementia diagnosed. Treatment strategies for Alzheimer’s disease or another type of dementia are ultimately the same for people with and without a history of traumatic brain injury. �

 

Alzheimer’s disease and other types of dementia which may occur as a long-term result of traumatic brain injury (TBI) are progressive health issues which worsen over time. As with all types of dementia, they can affect a person’s quality of life, shorten lifespan, and complicate the effort to manage other health issues effectively. However, because other types of dementia, such as CTE, are considerably new for researchers and healthcare professionals, clinical guidelines for diagnosis and treatment do not exist. Several research studies are underway to gain further insight into the patterns of TBI and Alzheimer’s disease which may be implicated in CTE and to develop strategies for prevention, diagnosis, and treatment. �

 

El Paso Chiropractor Dr. Alex Jimenez

As previously mentioned in the article above, Alzheimer�s disease and other types of dementia which may occur as a long-term result of traumatic brain injury (TBI) are progressive health issues which may ultimately worsen over time. As with all types of dementia, these can affect quality of life, shorten life span, and complicate the effort to manage other health issues effectively. It’s essential for patients and healthcare professionals to diagnose and treat a traumatic brain injury to prevent further health issues in the future, including Alzheimer’s disease and dementia. – Dr. Alex Jimenez D.C., C.C.S.T. Insight

 

According to research studies, TBI is ultimately associated with Alzheimer�s disease and other types of dementia. Doctors commonly characterize traumatic brain injury as mild, moderate, or severe depending on whether the previous traumatic event causes unconsciousness, how long it lasts, and other well-known symptoms. The scope of our information is limited to chiropractic, musculoskeletal and nervous health issues as well as functional medicine articles, topics, and discussions. To further discuss the subject matter above, please feel free to ask Dr. Alex Jimenez or contact us at 915-850-0900 . �

 

Curated by Dr. Alex Jimenez �

 


 

Additional Topic Discussion: Chronic Pain

 

Sudden pain is a natural response of the nervous system which helps to demonstrate possible injury. By way of instance, pain signals travel from an injured region through the nerves and spinal cord to the brain. Pain is generally less severe as the injury heals, however, chronic pain is different than the average type of pain. With chronic pain, the human body will continue sending pain signals to the brain, regardless if the injury has healed. Chronic pain can last for several weeks to even several years. Chronic pain can tremendously affect a patient’s mobility and it can reduce flexibility, strength, and endurance.

 

 


 

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Dr. Alex Jimenez utilizes a series of tests to help evaluate neurological diseases. The Neural ZoomerTM Plus is an array of neurological autoantibodies which offers specific antibody-to-antigen recognition. The Vibrant Neural ZoomerTM Plus is designed to assess an individual�s reactivity to 48 neurological antigens with connections to a variety of neurologically related diseases. The Vibrant Neural ZoomerTM Plus aims to reduce neurological conditions by empowering patients and physicians with a vital resource for early risk detection and an enhanced focus on personalized primary prevention. �

 

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If you are a patient of Injury Medical & Chiropractic�Clinic, you may inquire about XYMOGEN by calling 915-850-0900.

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