ClickCease
+1-915-850-0900 spinedoctors@gmail.com
Select Page

Imaging & Diagnostics

Back Clinic Imaging & Diagnostics Team. Dr. Alex Jimenez works with top-rated diagnosticians and imaging specialists. In our association, imaging specialists provide fast, courteous, and top-quality results. In collaboration with our offices, we provide the quality of service our patients’ mandate and deserve. Diagnostic Outpatient Imaging (DOI) is a state-of-the-art Radiology center in El Paso, TX. It is the only center of its kind in El Paso, owned and operated by a Radiologist.

This means when you come to DOI for a radiologic exam, every detail, from the design of the rooms, the choice of the equipment, the hand-picked technologists, and the software which runs the office, is carefully chosen or designed by the Radiologist and not by an accountant. Our market niche is one center of excellence. Our values related to patient care are: We believe in treating patients the way we would treat our family and we will do our best to ensure that you have a good experience at our clinic.


Spinal Neoplasms Diagnostic Imaging Approach Part I

Spinal Neoplasms Diagnostic Imaging Approach Part I

Metastatic Bone Disease (aka Mets) or “Secondaries.” Are the most common malignant bone neoplasms affecting the spine, aka spinal neoplasms (>70%) and the rest of the skeleton in adults.

  • 5-Primaries are m/c involved:
  • Breast (16-37%)
  • Lung (12-15%)
  • Thyroid (4%)
  • Renal (3-6%)
  • Prostate (9-15%)
  • Spine, pelvis, proximal femurs & proximal humeri are m/c affected in that particular order of frequency
  • Thoracic & upper Lumbar spine considered the m/c site of spinal Mets

Pathophysiology & Etiology of Metastasis

  • Malignant cells a very good at evading immune detection and elimination
  • They gain�access to circulation expressing Vascular Endothelial Adhesion Molecules (e.g., integrines & selectins)
  • Once reaching their target organs, malignant cells stimulate the production of various vasogenic growth factors and by exiting blood vessels invade their target tissues
  • Lung, Liver, and Bone are particularly at risk due to the character of their blood supply
  • Baston venous plexus-is a network of valveless freely communicating� veins connecting axial skeleton/meninges and proximal femurs/humeri with abdomino-pelvic and thoracic cavities
  • The risk of Mets is increased during daily variations in the intra-abdominal and intra-thoracic pressure

 

spinal neoplasms diagnostic imaging el paso tx.

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • In adults, the axial skeleton is involved in hematopoiesis, and it is particularly vulnerable to metastatic deposits via an abundant network of sinusoids within a spongy bone
  • The vast majority of bone Mets will be detected in the axial skeleton

Clinical Presentation

  • Back pain often mimicking “mechanical back pain” is the m/c and often misleading symptom
  • Chiropractors and other manipulators should be particularly aware of this dangerous pitfall.
  • Nocturnal pain or pain unresponsive to NSAID may be reported in more advanced cases
  • Advanced cases may also present with a neurological deficit due to pathologic vertebral fractures and spinal cord/nerves compression
  • Metastatic hypercalcemia may occasionally develop in severe cases and considered a medical emergency that potentially presents with confusion, muscle weakness, and renal signs
  • Imaging plays a significant role in the Dx and management of bone metastasis
  • Lab tests are of limited value, but hypercalcemia and alkaline phosphatase (Alk Phos) may be elevated
  • In some cases, a bone biopsy may be used to confirm bone Mets

When Bone Mets are Detected, Patients Prognosis is Significantly Worsened

  • Median survival:
  • Thyroid – 48 – months
  • Prostate – 40 – months
  • Breast – 24 – months
  • Renal Cell – may vary, can be as low as 6 – months
  • Lung – 6 – months

Imaging Diagnosis

  • Begins with radiography investigating a clinical complaint of back/bone pain
  • If radiographs are unrewarding or equivocal, unique imaging modalities are required
  • MRI may help to show marrow replacement by Mets foci but limited to specific regions
  • Tc99 radionuclide bone scan (scintigraphy) is considered one of the most sensitive and reliable imaging steps in evaluating bone Mets
  • Bone scintigraphy is good at detecting both lytic and blastic Mets
  • However, very aggressive/vascular osteolytic Mets and Multiple Myeloma often appear “cold” or photopenic on bone scan due to greater stimulation/activation of osteoclasts which “outpace” osteoblasts ability to uptake the radiopharmaceutical
  • CT scanning is an excellent modality to show bone destruction, but it is not widely used during bone Mets Dx especially if radiography, bone scintigraphy, and MRI provide adequate information about the process
  • CT scanning may be particularly helpful with delineation of pathological fractures

General Radiographic Features of Bone Mets

  • Osteolytic (lytic), osteoblastic (blastic) aka sclerotic Mets or misec Mets can be identified radiographically
  • However, it takes between 30-50% of lamella (cortical) bone and 50-75% of trabecular (cancellous) or spongy bone to be destroyed before it can be detected on plain film radiographs
  • This can make early radiographic detection of bone Mets very difficult, requiring particular imaging modalities (e.g., MRI)
  • Also, bowel gas/fecal matter and numerous soft tissue densities in the abdomino-pelvic and thoracic cavities may pose challenges of bone Mets detection
  • Different tumors often manifest with different metastatic appearance, depending on tumor activity and release of cytokines (IL6, IL11), endothelin 1 or other growth factors that will be responsible for either osteolytic, osteoblastic or mixed Mets
  • For example: purely lytic bone Mets are noted in Lung, Thyroid, and Renal cell CA (very vascular)
  • Breast CA may present with 60% of blastic Mets
  • Prostate CA presents with 90% of blastic Mets
  • Other blastic Mets may derive from urinary bladder, melanoma and GI adenocarcinomas
  • Sclerotic foci may also represent as previously treated primaries
  • Very vascular� Mets like Renal cell and Thyroid may present with markedly� lytic and expansile foci often called “blow out Mets.”
  • Mets found distal to elbows and knees (acro-metastasis) are commonly associated with Lung CA

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • PA chest view of a routinely screened patient with a known Hx of Prostatic adenocarcinoma
  • Note sclerotic lesion identified in the left posterior Rib 5
  • What imaging modality is required next?
  • Radionuclide bone scan should be suggested

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Multiple foci of high uptake of the Tc99 radiopharmaceutical
  • This is due to Mets and increased osteoblastic activity in the thoracic and lumbar spine, ribs and other sites of the skeleton

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Comparison of purely lytic (a and b) versus blastic (d) and mixed (c) Mets
  • What primaries to consider?

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Frog leg view of the hip
  • Clinical Dx: Prostatic adenocarcinoma
  • Note diffuse blastic Mets in the proximal femur
  • Hx: severe shoulder and arm pain unrelieved by rest
  • Rad DDx: Mets, Myeloma or less frequently Lymphoma
  • This classic DDx is used by the majority of Radiologists when aggressive osteolytic bone lesions are noted
  • The patient had a known Hx of Breast CA

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • A 51-year-old female with Breast CA
  • Large lytic destructive lesion in the distal femoral metaphysis characteristic of aggressive osteolytic Mets

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Sudden onset of severe leg pain and inability to stand in a 53-year-old female with Breast CA
  • Dx: Pathological fracture through the distal femoral shaft
  • Pathological Mets fractures in the spine and extremities are dreaded by most Oncologists due to higher association with severe complications and poor clinical prognosis

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Radiographic Dx of vertebral Mets should be suspected if a “missing pedicle sign” aka “winking owl sign” is noted
  • DDx: pedicle agenesis (above left) shows hypertrophy and sclerosis of a contralateral pedicle d/t increased mechanical stress
  • Pedicle Mets are often thought of as the m/c initial site of spinal Mets

 

spinal neoplasms diagnostic imaging el paso tx.

 

Vertebral Body Pathologic Fracture (VERTEBRA PLANA)

  • Isolated compression fracture at the T8 segment noted (above arrow)
  • The loss of the posterior and anterior height suggest an underlying pathologic condition for which the differential diagnosis includes:
  • Multiple myeloma
  • Metastatic Carcinoma
  • Other malignancy
  • Osteoporosis
  • Differentiating Pathological Fx of the vertebral body from an osteoporotic insufficiency Fx can be a significant challenge
  • Close inspection of the posterior body height is helpful but often not reliable
  • In metastasis, the posterior body is collapsed
  • In OSP, the posterior body may be maintained appearing more as anteriorly wedge fracture
  • MR imaging and/or radionuclide bone scan need to be performed

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • A skeletal radiographic survey may be used occasionally for the evaluation of bone Mets especially in well-established cases
  • It includes bilateral AP & lateral Thoracic and Lumbar views, AP pelvis, humeri, femurs, and the skull
  • Availability of special imaging has supplanted the use of skeletal radiographic survey
  • However, in a clinical practice skeletal radiographic study of Multiple Myeloma may still be used primarily if the diagnosis was previously established

 

spinal neoplasms diagnostic imaging el paso tx.

 

Technetium-99 (99mTc) bone scintigraphy is very sensitive and cost-effective study:

  • For the detection/localization of Mets and often an assessment of their biologic activity and response to treatment
  • This modality is a well-established part of the workup for known as well as unknown primaries
  • It may also help with determination of lesions that will be most accessible and easy to biopsy

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • When the burden of Mets is significantly high as shown in the case above
  • The radiotracer uptake is being almost entirely taken in by metastatic lesions
  • No material is left for the kidneys to excrete
  • This is known as a “super scan”

 

spinal neoplasms diagnostic imaging el paso tx.

 

  • Sagittal Lumbar and Lower Thoracic MRI. Multiple metastasis are noted on T1 (above right) and T2 (above left)� WI as hypointense foci of marrow replacement of the vertebral bodies in a patient with Hx of Prostate CA
  • MR imaging protocol with T1, T2, and T1+C gad can be used in many cases if x-radiography is unrewarding or questionable
  • �MRI can reveal bone marrow changes due to bone marrow replacement by Mets and surrounding edema
  • Typically blastic Mets appear as abnormally decreased signal intensity (hypointense) lesions on T1 and T2 pulse sequences
  • Purely lytic Mets often appear as hypo-intense on T1 and hype-intense on T2
  • Increased gadolinium uptake may also be evident on T1+C fat suppressed sequence d/t increased vascularity of malignant foci especially in very aggressive vascular neoplasms

Spinal Neoplasms

Spinal Arthritis Diagnostic Imaging Approach Part II

Spinal Arthritis Diagnostic Imaging Approach Part II

 

spinal arthritis el paso tx.

 

  • Spinal Arthritis
  • Ossification of Posterior Longitudinal Ligament (OPLL). Less frequent than DISH.
  • Greater clinical importance d/t spinal canal stenosis and cervical myelopathy
  • Asian patients are at higher risk
  • Both OPLL & DISH may co-exist and increase the risk of Fx
  • Imaging: x-rad: linear radioopacity consistent with OPLL
  • Imaging modality of choice: CT scanning w/o contrast
  • MRI may help� to evaluate myelopathy
  • Care: surgical with laminoplasty (above right image) that has been pioneered and advanced in the Far East

 

M/C Inflammatory Arthritis In Spine

 

spinal arthritis el paso tx.

 

  • Rheumatoid spondylitis (Rheumatoid arthritis) d/t inflammatory synovial proliferation pannus rich in lymphocytes, macrophages, and plasma cells
  • C/S RA may affect 70-90% of patients
  • Variable severity from mild to destructive disabling arthropathy
  • RA IN C/S m/c affects C1-C2 due to an abundance of rich synovial tissue
  • Typically infrequent in the thoracic/lumbar region
  • Sub-axial C/spine may be affected later due to facets, erosions, ligament laxity and instability showing “Stepladder” appearance
  • Clinically: HA, neck pain, myelopathy, etc. inc. Risk of Fx/subluxation. Any spinal manipulation HVLT ARE STRICTLY CONTRAINDICATED.
  • Rx: DMARD, anti-TNF-alfa, operative for subluxations, etc.

 

Rheumatoid Spondylitis C1-C2. Perform X-radiography initially with flexed-extended views. Note Dens erosion, C1-2 subluxation (2.5 mm) that changes on mobility

 

spinal arthritis el paso tx.

 

spinal arthritis el paso tx.

 

  • RA spondylitis: an erosion of the odontoid with the destruction of C1-C2 ligaments and instability
  • Stepladder aka Step-step sub-axial deformity d/t facets erosions and ligamentous destruction/laxity
  • MRI required to evaluate cord compression/myelopathy

 

spinal arthritis el paso tx.

 

  • Sagittal T2 WI MRI of pt with RA. Rheumatoid pannus formation is present at C1-2 (arrow) causing mild cord compression
  • RA pannus may develop early before frank x-radiography changes noted
  • Clinically: HA, neck pain, tingling in UE, positive Lhermitte phenomenon d/t cervical myelopathy

 

Operative Care of Rheumatoid Spondylitis and Its Complications

 

spinal arthritis el paso tx.

 

Seronegative Spondyloarthropathies

 

  • Ankylosing Spondylitis (AS)
  • Enteropathic Arthritis (EnA) (d/t IBD: Crohn’s & UC) identical to AS on imaging
  • Psoriatic Arthritis (PsA)
  • Reactive Arthritis (ReA)
  • All share the following features: m/c HLA-B27 marker, RF-, Sacroiliitis, Enthesitis, Ocular Involvement (i.e., conjunctivitis, uveitis, episcleritis, etc.)
  • AS & EnA are radiographically virtually indistinguishable, but EnA typically presents with less severe spinal changes than AS
  • Both PsA & ReA present with virtually identical spinal changes, but ReA typically affects the lower extremity compared to PsA affecting hands and feet

 

spinal arthritis el paso tx.

 

spinal arthritis el paso tx.

 

  • AS: likely autoimmune systemic inflammatory disease that targets SIJ, spinal facet joints annuls of the disc, rib joints and all spinal ligaments.
  • Key path feature: enthesitis.
  • Extraspinal features: uveitis, aortitis, pulmonary fibrosis, amyloidosis, cardiovascular disease.
  • M:F 4:1, age: 20-40 m/c. Clinic LBP/stiffness, reduced rib expansion <2 cm is > specific than HLA-B27, progressive kyphosis, risk of Fx’s.
  • Imaging steps: 1st step-x-rays to id. Sacroiliitis/spondylitis.�MRI & CT may help if x-rays are unrewarding.
  • Labs: HLA-B27, CRP/ESR, RF-
  • Dx: clinical+labs+imaging.
  • Rx: NSAID, DMARD, anti-TNF factor therapy
  • Key Imaging Dx: always presents initially as b/l symmetrical sacroiliitis that will progress to complete ankylosis. Spondylitis presents with continuous ascending discovertebral osteitis (i.e., marginal syndesmophytes, Romanus lesion, Anderson lesion), facets and all spinal ligament inflammation and fusion with a late feature of “bamboo spine, trolley track, dagger sign,” all indicating complete spinal ossification/fusion. Increasing risk of Fx’s.

 

spinal arthritis el paso tx.

 

Key Dx of Sacroiliitis

 

  • Blurring, cortical indistinctness/irregularity with adjacent reactive subchondral sclerosis initially identified primarily on the iliac side of� SIJs.
  • Normal SIJ should maintain a well defined white cortical line. Dimension 2-4 mm. May look incongruous d/t 3D anatomy masked by 2D x-rays.

 

spinal arthritis el paso tx.

 

Key Imaging Dx In Spine

 

  • Marginal syndesmophytes and inflammation at the annulus-disc (above arrows) at the earliest dx; by MRI as marrow signal changes on T1 and fluid sensitive imaging (above top images).
  • These represent enthesitis-inflammation that will ossify into bamboo spine.
  • Lig ossification: trolley track/dagger sign

 

spinal arthritis el paso tx.

 

  • AS in extraspinal joints: root joints, hips, and shoulders
  • Symphysis pubis
  • Less frequent in peripheral joints (hands/feet)
  • All seronegatives may present with heel pain d/t enthesitis

 

spinal arthritis el paso tx.

 

  • Complication: Above Carrot-stick/chaulk-stick Fx

 

spinal arthritis el paso tx.

 

  • PsA & ReA (formerly Reiter’s) present with b/l sacroiliitis that virtually identical to AS
  • In the spine PsA & ReA DDx from AS by the formation of non-marginal syndesmophytes aka bulky paravertebral ossifications (indicate vertebral enthesitis)
  • For a clinical discussion of Spondyloarthropathies refer to:
  • www.aafp.org/afp/2004/0615/p2853.html

 

Spinal Arthritis

Spinal Arthritis Diagnostic Imaging Approach Part I

Spinal Arthritis Diagnostic Imaging Approach Part I

Degenerative Arthritis

  • Spinal Arthritis:
  • Spondylosis aka Degenerative disease of the spine represents an evolution of changes affecting most mobile spinal segments beginning with:
  • Intervertebral disc (IVD) dehydration (desiccation) and degeneration aka Degenerative Disc Disease (DDD) with an abnormal increase in mechanical stress and degeneration of posterior elements affecting 4-mobile synovial articulations ( true osteoarthritis)
  • 2-Facets in the L/S & 2-Facets & 2-Uncovertebral joints in the C/S
  • Imaging plays a significant role in the diagnosis, grading, and evaluation of neurological complications (e.g., spondylotic myelopathy/radiculopathy)
  • X-radiography with AP, Lateral & Oblique spinal views provides Dx and classification of Spondylosis
  • MR imaging may help to evaluate the degree of neurological changes associated with degenerative spinal canal and neural foraminal stenosis
spinal arthritis el paso tx.
  • Spinal motion segment:
  • 2-adjacent vertebrae
  • IVD (fibrocartilage)
  • 2-facets (synovial)
  • Pathology: loss of disc height increases mechanical stress on mobile elements
  • Ligamentous laxity/local instability
  • Spinal osteophytes aka spondylophytes & bony facet/uncinate proliferation
  • Disc herniation and often disc-osteophyte complex
  • Ligamentum flavum “hypertrophy” or thickening due to buckling
  • Loss of normal lordosis with or w/o reversal or kyphosis
  • Vertebral canal & neural foraminal stenosis

Neutral lateral cervical radiograph: note mild to moderate disc narrowing and spondylophyte formation at C5-6 & C6-C7 (most common levels affected by cervical spondylosis). Straightening or flattening with mild reversal of cervical lordosis. Some mild facet proliferation is noted at the above levels

spinal arthritis el paso tx.
  • On radiographs: evaluate for disc height (mild, moderate or severe) loss
  • End-plate sclerosis & spondylophytes; mild, moderate or severe
  • Facet and uncinate irregularity, hypertrophy/degeneration; mild, moderate or severe
  • Note degenerative instability aka degenerative spondylolisthesis/retrolisthesis
  • Normal or lost lordosis vs. degenerative kyphosis
  • Key Dx: correlate with a clinical presentation: neck/back pain with or w/o neurological disturbance ( myelopathy vs. radiculopathy or both)
spinal arthritis el paso tx.
  • Uncinate processes undergo degeneration/proliferation resulting in uncovertebral arthrosis
  • Early findings present with mild bone proliferation along the cortical margin (white and black arrows) if compared to normal uncinate (orange arrow)
  • Later, more extensive bone proliferation extending into and narrowing vertebral canal and neural osseous foramina (IVF’s) may be noted. The latter may contribute to spinal/IVF stenosis and potential neurological changes
  • Posterior oblique views may help further
spinal arthritis el paso tx.
  • AP lower cervical (a) and posterior oblique (b) views
  • Note mild uncinated process proliferation with neural foraminal narrowing (arrows)
  • Typically if less than a third of IVF becomes narrowed, patients may present w/o significant neurological signs
spinal arthritis el paso tx.
  • Lumbar spondylosis is evaluated with AP and lateral views with additional AP L5-S1 spot view to examine lumbosacral junction
  • Typical features include disc height loss/degeneration
  • Intra-discal gas (vacuum) phenomenon (blue arrow) along with spondylophytes
  • Degenerative spondylolisthesis and/or retrolisthesis (green arrow) may follow disc and facet degeneration and can be graded by the Meyerding classification
  • In most cases, degenerative spondylolisthesis rarely progresses beyond Grade 2
  • Lumbar facet degeneration seen as bone proliferation/sclerosis and IVF narrowing
spinal arthritis el paso tx.
spinal arthritis el paso tx.
  • MR imaging w/o gad C is an effective modality to evaluate clinical signs of spondylosis & associated neurological complications with pre-surgical evaluation
  • Case: 50-y.o Fe with neck pain. Case b-45-y.o.M (top a b images). MRI reveals: loss of disc hydration or desiccation, spondylophytes and disc herniation w/o neurological changes
  • (Bottom images) Left: preoperative and right postoperative MRI slices of the patient presented with clinical signs of cervical spondylotic myelopathy. Note disc herniation, ligam flavum hypertrophy and canal stenosis (left)
spinal arthritis el paso tx.
  • Sagittal MRI slice of lumbar DDD manifested with disc desiccation and posterior herniation effacing thecal sac
  • Correlating sagittal and axial slices will be more informative to evaluate canal stenosis and potential degree of neurological involvement (above-bottom images)
spinal arthritis el paso tx.
spinal arthritis el paso tx.

Diffuse Idiopathic Skeletal Hyperostosis (DISH) aka Forestier disease

  • Flowing degenerative ossification of ALL
  • M/c Thoracic spine. 2nd m/c-cervical spine
  • Dx by imaging only. X-radiography is sufficient
  • CT w/o contrast helps with Dx of Fx
  • Men>women. Pts>60-y.o. Extensive DISH shows 49% association with type 2DM
  • Complications: Chalk (carrot) stick Fx. Unstable 3-column Fx requiring surgical fusion
  • Sagittal reconstructed CT scan slice in bone window
  • Chalk stick Fx at C5-C6 in the patient with DISH and OPLL

Spinal Arthritis

Spinal Trauma Imaging Approach to Diagnosis Part II

Spinal Trauma Imaging Approach to Diagnosis Part II

Hyperextension Injury

spinal trauma el paso tx.

  • Hangman’s Fx aka traumatic spondylolisthesis of C2 with a fracture of pars interarticularis or pedicles (unstable)
  • MVA is the most common cause
  • Mechanism: acute hyperextension of upper C/S similar to judicial hanging (never actually seen and most deaths are due to asphyxiation)
  • Secondary flexion may tear PLL and disc
  • Associated injuries: 30% have other c-spine fx especially Extension teardrop at C2 or C3 due to avulsion by ALL
  • Cord paralysis may only present in 25% due to bony fragments dissociation and canal widening
  • Hangman fx and extension teardrop
  • Cervical degeneration and previous fusion is a key predisposing factor due to the lack of mobility and suppleness, rendering C/S easy to fracture
  • Imaging: initial x-radiography then CT that helps to delineate another injury such as facet/pedicle Fx further. MRI may help if complicated by Vertebral A. damage
  • Management: if type 1 injury then closed reduction and rigid collar for 4-6 weeks, halo bracing if type 2 (>3-5mm displacement) Fx/instability, anterior or posterior spinal fusion at C2-3 if type 3 Fx (>5-mm displacement)

 

spinal trauma el paso tx.

 

  • Extension teardrop Fx (stable) potentially unstable if put in extension
  • Avulsion of an inferior anterior body by ALL. More seen in elderly with superimposed C/S spondylosis
  • Key radiography: a smaller anterior-inferior body corner, no disruption of ligamentous alignment. Typically at C2 or C3 due to sudden hyperextension and ALL avulsion
  • Complication: central cord syndrome (m/c incomplete cord injury) esp. in superimposed spondylosis and canal stenosis by the laxity of ligamentum flavum and osteophytes
  • Management: hard collar isolation

 

spinal trauma el paso tx.

 

Vertical (axial) Compression Injury

  • Jefferson Fx (named after British neurosurgeon who defined it) (unstable but neurologically intact Fx) 7% of all C/S injuries. Stability is dependent if the transverse ligament is intact or torn, which can be noted by overhanging of C1 lateral masses over C2 >5-mm combined (left image)
  • Mechanism: C1 compression (e.g., diving into shallow waters) causing burst Fx-classically 4-parts of the anterior and posterior arch of C1. Variations exist.
  • Complications: 50% show other C/S Fx, 40% show Odontoid C2 Fx esp. if extension and axial loading occur

 

spinal trauma el paso tx.

 

  • Imaging: x-radiography followed by CT scanning to evaluate subaxial injury and complexity of C1 injury. Note Jefferson Fx with pillar and transverse foramina fx requiring posterior occipital-cervical fusion (below right image).
  • Management: rigid collar immobilization if the transverse ligament is intact. Halo brace or fusion if the transverse ligament is ruptured

 

spinal trauma el paso tx.

 

Cervical Injuries With Variable Mechanisms of Trauma

  • Odontoid process fractures:
  • These occur�with a variety of mechanisms, flexion, extension, lateral flexion. Elderly with superimposed spondylosis are at higher risk.
  • Anderson & D’Alonzo classification (below). Type 2 is the most common and most unstable. Type 3 has the best chance of healing d/t more massive bleed into C2 body and better healing potential.
  • Imaging: x-radiography can miss some Fx. CT scanning is essential.
  • On x-radiography note tilting of the Dens on lateral and APOM views. CT will reveal the injury and classify it.
  • Complications: cord injury, non-union

 

spinal trauma el paso tx.

 

  • CT scanning: type 2 odontoid fracture (unstable)
  • Management: type 1 (alar ligament avulsion) most stable�observed and treated with rigid collar.
  • In young patients, Halo brace is used to treat type 2
  • Older patients do not tolerate Halo
  • Operative C1-2 fusion if unstable is Dx and cord signs or other complicating factors are present

 

spinal trauma el paso tx.

 

Normal Radiographic Variants & Anomalies Simulating Pathology

  • Pediatric spine appears different especially in children younger than 10-years old.
  • Normal variations; ADI 5-mm and may increase or decrease on flexed/extended views by 1-2-mm
  • C2-3 may appear as pseudo-subluxation due to normal ligamentous laxity in children (below arrow)
  • Pediatric vertebral bodies usually are narrower and anteriorly wedged due to the presence of cartilaginous tissue
  • APOM view appears different in children, and some asymmetry of C1 articular masses is normal (below top image) and should not be confused with Jefferson Fx
  • In adults, any asymmetry or “overhanging” of C1 articular masses is pathological and may indicate Jefferson fx

 

spinal trauma el paso tx.

 

  • Standard ossification centers of the Atlas synchondrosis in children should not be mistaken for fractures

 

spinal trauma el paso tx.

 

  • Persistent ossiculum terminal of Bergman is a typical variant/anomaly of tenacious un-united ossification center and should not be confused with type odontoid fx
  • Os odontoideum
  • Un-united growth center that currently considered as an un-noticed injury that disturbed normal growth in a child younger than 5-years-old
  • It may be a cause of C1-2 instability and should be evaluated with flexed and extended cervical views
  • Should not be confused with type 2 Dens fracture because it typically more demonstrates greater mineralization of bone

 

spinal trauma el paso tx.

 

  • Incomplete bilateral agenesis of the C1 posterior arch
  • Anomalous closure of C1 posterior arch
  • Should not be confused with a fracture
  • However, local or cord symptoms may develop after trauma in some cases
  • Relatively rare anomaly developing due to failed chondrogenesis and ossification of posterior ossification centers of the Atlas

 

spinal trauma el paso tx.

 

  • Patients with Down syndrome may suffer from increased ligamentous laxity and other abnormalities
  • Increased risk of subluxation at C1-2

 

spinal trauma el paso tx.

 

  • Burst Fx (unstable) 2-columns are damaged
  • Mechanism: axial loading with frequent flexion after falls and MVAs
  • The thoracolumbar region is the most vulnerable due to the increased fulcrum of motion
  • Key radiography: acute compression fracture and�collapse of body height, retropulsion of posterior body and acute kyphotic deformity on the lateral view
  • On the frontal view: interpedicular widening (below yellow arrow), regional soft tissue swelling (below green arrow)

 

spinal trauma el paso tx.

 

  • Imaging: x-radiography should be followed by CT scanning w/o contrast
  • MRI if neurologically unstable due to cord or conus injury
  • Complications: cord damage by acutely retropulsed bone fragments
  • Management: non-operative if neurologically intact and <50% body retropulsed with minimal kyphosis
  • Operative (fusion) if 50% or more body retropulsed, laminar/pedicle Fx, neuro compromised

 

spinal trauma el paso tx.

 

18-Year Old Female Following Trampoline Accident

  • AP & lateral L/S views
  • Note acute compression fracture, a vertebral body extending to posterior elements
  • Widening of the inter-spinous distance between T11-T12 (below arrow)
  • Radiolucent fracture line is seen through the T12 body on the AP projection
  • CT scanning was performed

 

spinal trauma el paso tx.

 

  • Sagittal reconstructed Thoracic and Lumbar CT slices in bone window
  • Note acute compression fracture, the T12 body extending into pedicle and lamin
  • Dx: Chance fracture of T12
  • MR imaging was performed

 

spinal trauma el paso tx.

 

  • T2 Wl sagittal MRI
  • Findings: acute compression fracture T12 body extending to posterior elements causing rapture of interspinous and flavum ligaments
  • Mild compression of the distal cord above the conus is noted with a minimal signal abnormality
  • Dx: Chance fracture

 

spinal trauma el paso tx.

 

  • Chance Fx aka (Seatbelt Fx) – is a flexion-distraction injury (unstable)
  • M/C in lower thoracic-upper lumbar
  • All 3-columns fail: column 3 torn by distraction, columns 1 and 2 fail on compression (Denis classification)
  • Causes: MVA, falls
  • Imaging: initial x-radiography should be followed by CT scanning w/o contrast to assess bone fragments retropulsion/canal compression. MRI may help to evaluate potential cord damage and ligaments tearing
  • Management: non-operative immobilization if neuro intact
  • Operative decompression and fusion

 

spinal trauma el paso tx.

 

Spinal Trauma Imaging Approach

Resources:

Spinal Trauma Imaging Approach to Diagnosis Part I

Spinal Trauma Imaging Approach to Diagnosis Part I

Imaging Diagnosis Management:

  • Cervical spinal trauma & radiographic variants simulating disease
  • Cervical spine
  • Arthritis
  • Neoplasms
  • Infection
  • Post-Surgical cervical spine

 

spinal trauma el paso tx.

 

  • Cranio-cervical and upper cervical stability is dependent on transverse, superior and inferior bands of the C1-C2 ligament, alar ligaments, along with a few other ligaments

 

spinal trauma el paso tx.

 

spinal trauma el paso tx.

 

Cervical Trauma

  • The C/S is vulnerable to injury. Why?
  • Stability has been sacrificed for greater mobility
  • Cervical vertebrae are small and interrupted by multiple foraminae
  • The head is disproportionately heavy and acts as an abnormal lever especially when forces act against a rigid torso
  • Additionally, C/S is prone to degeneration which makes it more vulnerable to trauma
  • In young children, ligaments are more luxed vs. disproportionately large head size
  • In children, the fulcrum of movement is at C2/3 thus making injuries more common in the upper C/S and craniocervical junction. In children, S.C.I.W.O.R.A. may occur when no evidence of fracture present
  • In adults, the fulcrum of movement is at C5/6 thus making lower C/S more vulnerable to trauma especially during extremes of flexion
  • Cervical Trauma categorized according to mechanisms of injury (Harris & Mirvis classification)

 

Hyperflexion Injury: Stable vs. Unstable

  • Flexion teardrop Fx (most severe fracture, unstable)
  • Bilateral facet dislocation (severe injury w/o fracture, unstable)
  • Anterior subluxation (potentially unstable) can be very subtle injury
  • Clay Shoveller Fx (lower C/S SP avulsion, stable)
  • Simple wedge compression (most benign Fx, stable)
  • Hyperflexion-rotation with unilateral facet dislocation
  • Obtain a thorough history
  • Perform physical exam including a neurological exam
  • Consider NEXUS criteria (National Emergency X-radiography Utilization Study)

 

Imaging Techniques:

  • Begins with x-radiography especially in cases with no significant neurological compromise
  • Clear neutral lateral view first
  • If x-radiography is unrewarding but high probability of severe trauma and neurological deficit present, CT scanning w/o contrast is required
  • Consider CT scanning in patients with pre-existing changes: advance spondylosis, DISH, AS, RA, post-surgical spine, congenital abnormalities (Klippel-Feil syndrome, etc.)

 

Vertical compression:

  • Jefferson aka burst Atlas Fx (unstable especially if the Transverse ligament is torn, cord paralysis in 20-30% only)
  • Why? Due to fragments dissociation and canal widening
  • Burst Fx of the Thoracic or Lumbar spine (unstable, cord paralysis may occur)

 

spinal trauma el paso tx.

 

How to Assess Spinal Radiographs in Trauma Cases:

  • Construct 5-lines on the lateral view
  • Note if facets are well-aligned and symmetrical
  • Ensure symmetry of the disc height
  • Note any widening or fanning of the inter-spinous distance
  • Carefully examine prevertebral soft tissues
  • Evaluate atlanto-dental interval (ADI)

 

spinal trauma el paso tx.

 

  • In cases of trauma, evaluate and clear neutral lateral first
  • Do not perform flexed and extended views in acute cases before x-rays or CT scanning exclude significant instability
  • Pay extra attention to prevertebral soft tissues
  • If thicker than normal limits, consider severe post-traumatic bleed
  • Subtle asymmetry and widening of posterior disc height and facets with inter-spinous fanning may be a key feature of significant tearing of posterior ligaments

 

spinal trauma el paso tx.

 

Hyperflexion Injuries (M/C Mechanism)

  • More frequent in sub-axial C/S C-3-C7)
  • Unstable injuries:
  • Flexion teardrop fracture (M/C C5 & C6) v. unstable
  • Key rad features:
  • Large “teardrop” triangular anterior body fragment
  • Fanning of the SPs, posterior disc and facet widening indicating tearing of major spinal ligaments and instability
  • A posterior shift of the vertebral body fracture suggests direct anterior cord/vessels compression
  • Bulging prevertebral soft tissue >20-mm at C6-7
  • 80% of cases may be paralyzed on the spot or develop significant paralysis soon after

 

spinal trauma el paso tx.

 

Acute Neck Trauma. What are the vital radiographic features? What is the diagnosis?

 

spinal trauma el paso tx.

 

  • CT scanning w/o contrasts with sagittal reconstruction. Note C7 Flexion teardrop Fx.
  • CT may help with further delineation and preoperative planning
  • May follow with MR imaging and evaluation of the neurological injury

 

spinal trauma el paso tx.

 

  • Fluid sensitive (T2) sagittal MRI slice of Flexion teardrop fracture at C4 and possibly C5
  • Note high signal intensity lesion in the cord and surrounding ligaments indicating cord edema and ischemia
  • Management: neurosurgical with spinal fusion
  • Complications:
  • Quadriplegia/paraplegia
  • Respiratory complications
  • Disability, changes in the quality of life
  • Decreased life expectancy

 

spinal trauma el paso tx.

 

  • Bilateral facet dislocation (unstable)
  • Mechanism: Flexion-distraction injury
  • Key radiography: anteriorly displaced body 50% or more
  • Facets override and locked (can be perched left image)
  • Major tearing of ligaments
  • Chances of severe cord compression and paralysis
  • Patients with ligaments laxity and degenerative changes are at higher risk
  • Initial x-radiography is the first step

 

spinal trauma el paso tx.

 

CT scanning w/o Contrast is Crucial:

 

spinal trauma el paso tx.

 

  • Further delineation of this injury
  • Facet fractures, pedicle fracture
  • Management planning

Sagittal fluid sensitive MRI of bilateral C5 facet dislocation, sizeable ischemic cord injury, and posterior soft tissue injuries

 

spinal trauma el paso tx.

 

  • Management:
  • X-radiography, then CT scanning then immediate closed reduction (esp. if the patient is conscious)
  • Followed in some more complicated cases by MRI and then surgical care
  • If the patient is awake and neurologically stable, CT and closed reduction are adequate
  • Complicated cases and failed closed reduction may require surgical stabilization
  • Complications: spinal cord injury and paralysis
  • Delayed ligamentous laxity and instability

 

spinal trauma el paso tx.

 

    • Unilateral facet dislocation (flexion-rotation injury) less severe than bilateral dislocation
    • Most commonly missed unstable cervical injury on x-radiography
    • Key rad features: body anteriorly translated 25% facets appear misaligned and blurred, SPs rotated on frontal views
    • Clinically may be presented as one-sided radiculopathy esp. C6 or C7
    • CT scanning is required to evaluate further facet/pedicle fractures
    • Pre-reduction evaluation and care planning
    • Management: closed reduction esp. in a conscious patient
    • Complications: acute disc herniation/retropulsion, ligamentous laxity, neurological injury

Spinal Trauma Imaging Approach

Resources:

 

Introduction To Medical Imaging Conventional Radiography

Introduction To Medical Imaging Conventional Radiography

  • Conventional Radiography is 2-D imaging modality
  • It is required to perform minimum 2-views orthogonal to each other:
  • 1 AP (Anterior to Posterior) or PA (Posterior to Anterior)
  • 2 Lateral
  • Supplemental views: Oblique views etc.
  • Skeletal radiographs typically use AP & lateral views
  • Chest radiographs and Scoliosis imaging in children will usually use the PA technique
  • Exceptions for PA chest views: patients unable to cooperate (severely ill or unconscious patients)
  • X-rays are a form of electromagnetic energy (EME) similar to light photons or other sources
  • X-rays are a form of man-made radiation
  • Ionizing effect of x-rays process of removal of atomic electrons from their orbits
  • Two basic types of ionizing radiation:
  • Particle (particulate) radiation produced by alpha & beta particles that are the result of radioactive decay of different materials
  • Electromagnetic Radiation (EMR) produced by x-rays or gamma rays called photons
  • The energy of EMR depends on its wavelength
  • Shorter wavelength corresponds to higher energy
  • The energy of EME is inversely related to its wavelength
imaging and diagnostics el paso tx.

X-ray Properties

  • No charge
  • Invisibility
  • Penetrability of most matters (esp. human tissues) depends on “Z” (atomic number)
  • Making compounds fluoresce and emit light
  • Travel at the speed of light
  • Ionization and biologic effect on living cells

The Imaging System

  • X-rays are produced by an imaging system ( x-ray tube, operator’s console, and high voltage generator)
  • X-ray tube composed of (-) charged cathode and (+) charged anode enclosed in the evacuated class envelope and housed in the protective coat of metal
  • A Cathode made up of filament wire embedded within the focusing cup to give electrostatic focus to electrons’ cloud
  • Filament wire of heat resistant thorium tungsten metal of high melting point (3400 C) that “boils off” electrons during thermionic emission
  • Focusing cup polished nickel (-) charged that�accommodated� the filament to electrostatically repulse the electrons and confines them to the focal spot of the anode disc where x-rays are produced
imaging and diagnostics el paso tx.
  • Anode (+) charged target for electrons to interact at the focal spot
  • Conducts electricity
  • Rotates to dissipating heat
  • Made of tungsten to resist heat
  • Anode has a high atomic number to produce x-rays of very high efficiency at the focal spot
  • There are 2-focal spots large and small, each corresponding to cathode’s filament size (small vs. large) that depends on the magnitude of current in the cathode dictated by a radiographic study of larger or smaller body parts
  • It is known as the dual focus principle
imaging and diagnostics el paso tx.

When Electrons are emitted from the cathode as the cloud, they slam into the Anode’s focal spot resulting in 3 man events

  • Production of heat (99% outcome)
  • Production of Bremsstrahlung (i.e., breaking radiation) x-rays that represent the majority of x-rays within the x-ray emission spectrum
  • Production of Characteristic x-rays very few in the emission spectrum
imaging and diagnostics el paso tx.
  • Newly formed x-rays at the anode are of different energies
  • Only need high energy or “hard” x-rays to perform the radiographic study
  • Before x-rays exiting the tube we need to remove weak or low energy photons, i.e., “harden the beam.”
  • Added tube filtration in the form of aluminum filters is used that removes at least 50% of the “unfiltered” beam thus minimizing the patient’s radiation dose and maximizing image quality
imaging and diagnostics el paso tx.

High Voltage Generator

  • X-ray production requires an uninterrupted flow of electrons to the anode
  • Regular electricity supplies AC power with sinusoidal currents of “peaks and drops.”
  • In the past, single-phase high voltage generators would convert AC power into a half, or full wave rectified supply with a measure in the thousands of volts delivered with a “voltage ripple” or peaks of high voltage. Therefore, a term kilo voltage peaks (kVp) was used
  • Modern generators provide “uninterrupted” flow of electrical potential to the x-ray tube eliminating “voltage ripples” thus referred to as kilovoltage kV without “peaks.”

When x-rays interact with the patient’s tissued 3 events will occur

  1. X-rays will pass through without interaction and “expose” the image receptor
  2. Photoelectric interaction/effect (PE) comparatively lower energy x-rays will be absorbed/attenuated by the tissues
  3. Compton scatter x-rays are “bounced off” to form scatter, contributing no useful information to the film and lower image contrast while potentially giving unnecessary radiation dose to staff
  • The final image is the product of all three types of interactions known as
  • Differential absorption of x-ray photons – the result of photons’ absorption via PE, Compton scatter and x-rays passing through the patient
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
  • Compton scatter probability decreases with an increase in x-ray energy compared to PE effect
  • Compton effect probability does not depend on the atomic number (Z)
  • An increase of total mass density (thick vs. thin) will increase Compton and PE interaction
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.

What cells in the body are considered most vulnerable and most resistant to radiation?

  • Cells that are rapidly dividing and not terminally differentiated, epithelial cells, etc. are more radiosensitive
  • Bone marrow cells (stem cells) & lymphocytes are very radiosensitive
  • Muscle & and nerve cells are terminally differentiated and are less sensitive to radiation
  • Aged (senescent cells) vs. immature fetal cells are more vulnerable to radiation
  • However, following low dose radiation in most healthy individual cells will be able to repair likely without any long-lasting changes
imaging and diagnostics el paso tx.
  • Pregnancy & radiation initial 6-7 weeks are the most vulnerable
  • Do not use routine (non-emergent) radiographic examinations in pregnancy
  • Apply 10-day rule establish that radiographs can only be obtained during the initial ten days from the onset of the last menstrual cycle
  • Radiographic imaging of children:
  • If clinically possible use non-ionizing forms of medical imaging (e.g., ultrasound)
imaging and diagnostics el paso tx.
imaging and diagnostics el paso tx.

Non-axial imaging studies that use x-ray photons:

  • Conventional radiography
  • Fluoroscopy
  • Mammography
  • Radiographic angiography (currently less often used)
  • Dental imaging
  • Cross-sectional imaging using x-ray photons: Computed Tomography

Indication and Contraindication for conventional radiographic imaging

  • Advantages of Radiography: widely available, inexpensive, low radiation burden, the first step in imaging investigation of most MSK complaints
  • Disadvantages: 2D imaging, relatively lower diagnostic yield during an examination of soft tissues, numerous artifacts, and dependence on correct radiographic factors selection, etc.

Indications:

  • Chest: initial assessment of lung/intrathoracic pathology. Potentially determines or obviates the need for chest CT scanning. Pre-surgical evaluation. Imaging of pediatric patients due to extremely low radiation dose.
  • Skeletal: to examine the bone structure and diagnose fractures, dislocation, infection, neoplasms, congenital bone dysplasia, and many forms of arthritis
  • Abdomen:�can assess acute abdomen, abdominal obstruction, free air or free fluid within the abdominal cavity, nephrolithiasis, evaluate placement of radiopaque tubes/lines, foreign bodies, monitor resolution of postsurgical ileus and others
  • Dental: to asses common dental pathologies
The Knee

The Knee

The Knee | MRI may be requested for:

  • Ligament injuries
  • Meniscal tears and degeneration
  • Rheumatoid arthritis
  • Osteochondral fractures
  • Tendon disruptions

Bones & Cartilage Of The Knee

The knee joint is the largest, most complicated, and most vulnerable joint in the body, as it does not have a stable bony configuration. It consists of the tibiofemoral and patellofemoral articulations, which include the femur, tibia, and patella. The knee is a synovial joint that is enclosed by a ligament capsule. The capsule contains synovial fluid that keeps the joint lubricated (Figure 82). The knee provides flexible movement, but must also bear large weight and pressure loads. During walking, the knees support 1.5 times your body weight. When climbing stairs, they support 3-4 times your body weight. When squatting, your knees support 8 times your body weight.

the knee

Figure 82. Anatomy of the knee.

The tibiofemoral articulation is a modified hinge joint that allows bending and straightening, but also allows for slight rotation. This articulation consists of the lateral and medial condyles of the femur resting on the lateral and medial aspects of the tibial plateau. The femoral condyles make up the distal portion of the femur, which is expanded in order to assist with weight distribution at the knee joint. The medial femoral condyle is typically larger and rounder. The condyles are united anteriorly to provide the articular surface for the patella, but they are separated posteriorly by the intercondylar notch. This notch, or fossa, is the attachment site for the cruciate ligaments, the ligaments of Humphrey and Wrisberg, and the frenulum of the patellar fat pad. A large part of the posterior distal femur is called the popliteal surface. This area is covered by fat, which separates it from the popliteal artery. The medial and lateral edges of the popliteal surface are attachment sites for muscles. Superior to the femoral condyles are the epicondyles, which are the attachment sites for muscles, tendons, and capsular ligaments. The medial epicondyle is the attachment site for the medial (or tibial) collateral ligament (Figure 83). The lateral femoral epicondyle is the attachment site for the lateral (or fibular) collateral ligament, as well as the tendon of the popliteus muscle, fibers of the iliotibial tract, and the lateral capsular ligament. Superior and posterior to the epicondyles is the most distal extent of the linea aspera, the bony ridge of the femur.

The tibia is the distal portion of the tibiofemoral articulation at the knee. The tibia is the second longest bone in the body, ranked just behind the femur. Its proximal end is flattened and expanded to provide a larger surface for the body weight that is transmitted through the femur. Like the femur, the proximal tibia has medial and lateral condyles. The medial condyle is larger, and somewhat flattened where it contacts the medial meniscus. The lateral condyle has a circular look to its femoral articular surface. The lateral tibial condyle articulates with the head of the fibula posteriorly, which is as close as the fibula comes to any involvement in the knee joint. Both the medial and lateral condyles rise in the center of the superior aspect of the tibia to form the intercondylar eminence. Posterior to this eminence are the attachments sites for the posterior horns of the medial and lateral menisci, which will be discussed with the ligaments of the knee. The medial and lateral tibial condyles, and the area of the intercondylar eminence are often grouped together and referred to as the tibial plateau (Figure 84). This is a critical weight-bearing area, and greatly affects the stability of the knee joint. The tibial tuberosity (or tubercle) is located on the anterior surface of the proximal tibial shaft. It has a smooth upper portion, and a roughened lower portion, which is the insertion site for the patellar tendon. The lateral side of the tibial tuberosity has a ridge for the attachment of fibers from the iliotibial tract. This is the strongest direct attachment site for the iliotibial tract. The IT tract, or band, helps in limiting lateral movement of the knee.

the knee

Figure 84. Tibial plateau.

the knee

Figure 83. Tibiofemoral anatomy.

 

 

 

 

 

 

 

 

 

 

The patella is the third bone involved in the knee joint, specifically in the patellofemoral articulation. Patella means �little plate� in Latin, which describes the look and function of this sesamoid bone. The patella develops in the tendon of the quadriceps femoris muscle (Figure 85). It moves when the leg moves, and protects the knee joint by relieving friction between the bones and muscles when the knee is bent or straightened. The patellofemoral joint is a saddle-type synovial joint, allowing the patella to glide along the bottom front surface of the femur between the femoral condyles in the patellofemoral groove. Ossification of the patella is typically completed in females by age 10, and in males between the ages of 13-16. If the patella has more than one ossification center, and the additional center does not fuse, it is termed a bipartite patella (Figure 86).

the knee

Figure 86. Bipartite patella.

the knee

Figure 85. Patella location.

 

 

 

 

 

 

 

 

 

 

 

Articular, or hyaline, cartilage covers the ends of the bones involved in any joint. In the knee joint, this includes the distal end of the femur, the proximal end of the tibia, and the posterior aspect of the patella (Figure 87). In larger joints, this cartilage is approximately �� thick. Articular cartilage is white, shiny, rubbery, and slippery, enabling surfaces to slide against one another without damage. Articular cartilage is very flexible, due in part to its high water content, which also makes it highly visible on MRI. In contrast to the bones that it covers, articular cartilage has almost no blood vessels, so it is not good at repairing itself. Bones, on the other hand, have numerous blood vessels, and are good at self-repair.

the knee

Figure 87. Articular cartilage.

Another type of cartilage is found between the femur and tibia- the fibrous cartilage that makes up the medial and lateral menisci. The menisci, also referred to as �articular disks�, wrap around the round ends of the femur to fill the space between the femur and tibia (Figure 88). Since the menisci are more fibrous in composition, they have tensile strength and can resist pressure. They can help spread the force from our body weight over a larger area. By helping with weight distribution, the menisci protect the articular cartilage on the ends of the bones from excessive forces. The menisci are fashioned to be thicker on their outsides, creating a shallow socket on the tibial surface. They act like a wedge on the rounded distal portion of the femur, improving the overall stability of the knee joint by preventing any �rolling� of the femur. Despite how strong they sound, the menisci can crack or tear when the knee is forcefully rotated or bent. The medial meniscus is fused with the medial collateral ligament, so it is less mobile than the lateral meniscus. It is often injured when the anterior or posterior cruciate ligaments are injured. The inner 2/3 of the medial meniscus receives a limited blood supply, so the entire meniscus is usually slow to heal. The lateral meniscus suffers from fewer injuries than the medial meniscus. Meniscal tears are one of the most common causes of knee pain, with suspected meniscal tears the most common indication for an MRI of the knee joint.

the knee

Figure 88. Superior view of menisci of right knee.

Symptoms that might indicate a problem with the bones of the knee joint include locking of the joint, the knee giving way, crackling or grinding felt in the joint, and pain and swelling. Locking of the joint can be indicative of a �loose body� (bone, cartilage, or foreign object) in the joint space, which can often be removed through arthroscopy (Figure 89). A knee that gives way can indicate that the patella is out of the patellofemoral groove, which leaves the knee unstable. Crackling and grinding at the joint can result from degenerative arthritis or osteoarthritis, as well as from a dislocating patella. An increase in pain with activity can occur due to a stress fracture or bone fracture. One of the pathologic conditions that can affect the bones of the knee joint is osteochondritis dissecans, which can affect the distal femur, and was discussed previously with the femur anatomy. Various types of arthritis manifest in the bones of the knee joint, including osteoarthritis, infectious arthritis, and rheumatoid arthritis. Chondromalacia patella, also known as patellofemoral syndrome or �runner�s knee� results from an irritation of the undersurface of the patella (Figure 91). If the patella is not tracking correctly in the patellofemoral groove, the articular cartilage may rub against the knee joint (Figure 90). The cartilage degenerates, and becomes irritated and painful. This condition is most common amongst young, healthy athletes, especially females and runners that are flat-footed. Treatment is typically rest and physical therapy to stretch and strengthen the quads and hamstrings. If surgery is required, it may be to perform a �lateral release�, as the abnormal tracking of the patella can cause a tightening of the lateral tissues of the knee. The lateral release procedure cuts the tight tissues, so the patella can return to its normal position and tracking. Osgood-Schlatter disease involves the anteriorly located tibial tuberosity, and the patellar tendon that inserts on that tuberosity (Figures 92, 93). This condition affects children during their growth spurts, and is typically found more in boys. During growth spurts, contractions of the quad muscle put additional stress on the patellar tendon at its attachment site on the tibial tuberosity. This can result in multiple subacute avulsion fractures and inflammation of the tendon. Excess bone growth occurs on the tuberosity, and a lump on the tuberosity can be seen and felt. This lump can become irritated and swollen, causing knee and leg pain. This condition is typically worsened with running, jumping, and climbing stairs. Osgood-Schlatter usually resolves with rest, ice, compression and elevation, as well as maturity of the youngster�s skeleton.

Figure 89. Intraarticular loose body.

 

Figure 90. Patellofemoral groove.

Figure 91. Patellofemoral syndrome or �runner�s knee�.

 

 

 

 

 

 

 

 

Figure 92. Xray displaying Osgood-Schlatter disease.

 

Figure 93. MRI displaying Osgood- Schlatter disease.

 

Ligaments Of The Knee

Ligaments are the tough bands of tissue that connect bones. They are considered to be �viscoelastic�, meaning they can gradually lengthen under tension, but return to their original shape when the tension is removed. However, if they are stretched for a prolonged period of time, or past a certain point, the ligaments cannot retain their original shape, and may eventually tear or snap. This is one of the reasons that a dislocated joint should be re-located as quickly as possible. If the ligaments lengthen, they leave the joint weakened and prone to future dislocations. Controlled stretching exercises to lengthen ligaments, and make the joints more supple, are part of the daily routines of athletes, gymnasts, dancers, etc. Damaged ligaments can lead to unstable joints, wearing of the cartilage, and eventually osteoarthritis. The numerous ligaments of the knee joint are the most important structures in controlling stability of the knee. Many of these ligaments were mentioned in the femur anatomy section, as they have attachments on the distal femur. The more important ligaments will be reviewed here in greater detail, in regards to their functions in the knee joint. The main intracapsular ligaments are the anterior and posterior cruciates (Figures 94, 95). Intracapsular ligaments are not very common in synovial joints. They provide stability, but permit a larger range of motion as compared to capsular or extracapsular ligaments. The anterior cruciate ligament (ACL) stretches from the lateral femoral condyle to the anterior intercondylar area of the tibia, preventing the tibia from being pushed too far anterior relative to the femur. It is the more commonly injured of the cruciate ligaments, and can be torn during twisting and bending of the knee. Women are at higher risk for ACL ruptures due to the facts that the maximum diameter of the intercondylar fossa is in its posterior aspect (the ACL attaches anteriorly), and the overall width of the intercondylar fossa is smaller in females. The posterior cruciate ligament (PCL) stretches from the medial femoral condyle to the posterior intercondylar area of the tibia, preventing posterior displacement of the tibia relative to the femur. It is the stronger of the two cruciate ligaments, and is injured less frequently; however, it can be injured from direct force or trauma. The menisci are also considered to be intracapsular structures, with connections to ligaments inside and outside the joint capsule. Two of their intracapsular ligaments are the anterior and posterior transverse meniscomeniscal ligaments. They attach the medial and lateral menisci to each other at their anterior and posterior aspects. Posterior transverse meniscal ligaments are very rare- only 1-4% of knees will have them. Two additional intermeniscal ligaments are the medial and lateral oblique meniscomeniscal ligaments (Figure 96). Their names describe their anterior horn attachment sites; they attach on the posterior horn of the opposite meniscus (i.e. medial oblique meniscomeniscal attaches to the anterior horn of the medial meniscus and posterior horn of the lateral meniscus). The oblique meniscomeniscal ligaments both traverse the intercondylar notch, and pass between the anterior and posterior cruciate ligaments (Figure 97).

the knee

Figure 94. Cruciate ligaments and menisci.

the knee

Figure 95. Posterior view of cruciate ligaments of left knee.

the knee

Figure 96. Axial fatsat T2 FSE image with arrow indicating
oblique meniscal ligament coursing from anterior horn of
medial meniscus to posterior horn of lateral meniscus.

the knee

Figure 97. Sagittal dual-echo T2 through the intercondylar notch at the level of the posterior cruciate ligament (curved arrow); thin linear structure of low signal intensity inferior to PCL represents the oblique meniscomeniscal ligament (straight arrow); sometimes misinterpreted as displaced meniscal fragment.

 

The medial (or tibial) collateral ligament is considered a capsular ligament, as it is part of the articular capsule surrounding the synovial knee joint. It acts as mechanical reinforcement for the joint, protecting the knee from valgus force, or being bent open medially due to stress on the lateral side of the knee. The medial collateral ligament (MCL) is one of the most commonly injured of all knee ligaments, occurring in all sports, in all ages, and often times with medial meniscal tears (Figures 98-101). It has both superficial and deep components. Fibers from the superficial portion of the MCL attach to the medial epicondyle of the femur and the medial tibial condyle. Fibers from the deep medial collateral ligament attach to the medial meniscus. Proximal to the attachment point, this ligament is referred to as the meniscofemoral ligament, as it attaches the medial meniscus to the medial aspect of the femur. Distal to the meniscal attachment, the ligament is referred to as the meniscotibial (or coronary) ligament, as it attaches the medial meniscus to the medial aspect of the tibia. The meniscofemoral and meniscotibial are also referred to as the meniscocapsular or medial capsular ligaments, as they play an important role in anchoring peripheral parts of the medial meniscus in the medial side of the knee. The meniscotibial ligament is typically injured more often than the meniscofemoral ligament. The meniscotibial ligament attaches to the tibia several millimeters inferior to the articular cartilage. Its job is to stabilize and maintain the meniscus in its proper position on the tibial plateau. Disruption of the meniscotibial ligament can result in a floating meniscus or meniscal avulsion, while the meniscofemoral ligament may not be affected. The deep medial collateral ligament is short, and tightens quickly with rotation motions. It is often damaged, along with the ACL, when the mechanism of injury involves tibial rotation. Diagnosis and surgical repair of the deep medial collateral ligament can be challenging.

the knee

Figure 98. Normal MCL is linear,
has low signal intensity.

the knee

Figure 99. Grade 1 sprain shows adjacent edema, no change in signal intensity of MCL.

the knee

Figure 100. Grade 2 sprain or partial tear shows increased edema,
abnormal signal intensity,
thickening or thinning of ligament.

the knee

Figure 101. Grade 3 involves complete disruption of ligaments or attachments.

 

In addition to fibers of the medial collateral ligament, the deep portion of the capsular compartment of the medial knee is the location of the medial knee�s posterior support. The posterior oblique ligament is attached proximally to the medially located adductor tubercle of the femur, and distally to the tibia and the posterior aspect of the knee joint capsule. If the posterior oblique is injured, it is usually torn from its femoral origin. The posterior oblique ligament provides static resistance to valgus loads as the knee moves into full extension, as well as dynamic stabilization to valgus forces (stress from lateral side) as the knee moves into flexion. It acts as an important restraint to posterior tibial translation in cases of posterior cruciate ligament injury. The posterior oblique ligament has three �arms�. Its superior capsular �arm� becomes continuous with the posterior knee capsule, and the proximal portion of the oblique popliteal ligament. The oblique popliteal ligament is also an important posterior stabilizing structure for the knee joint Figure 102). It extends from the posteromedial aspect of the tibia, running obliquely and laterally upward to insert near the lateral epicondyle of the femur.

the knee

Figure 102. Oblique popliteal ligament in posterior view of knee.

the knee

Figure 103. Medial (tibial) and lateral (fibular) collateral ligaments.

 

The lateral (or fibular) collateral ligament is considered an extracapsular ligament. It helps to provide joint stability and protects the lateral side of the knee from varus forces, or inside bending forces that are directed at the medial side of the knee. Injuries to the lateral collateral ligament are less common than injuries to the medial collateral, as the opposite leg can guard against medial forces that can lead to lateral collateral injuries. Injuries can occur in sports such as soccer and rugby, where the knee is extended and unprotected during running. The lateral, or fibular, collateral ligament stretches obliquely downward and backward, from the lateral epicondyle of the femur to the head of the fibula (Figure 103). It is not fused with the capsular ligament or with the lateral meniscus, so it has increased flexibility and decreased incidence of injury when compared to the medial collateral ligament. Similar to the medial meniscus, the lateral meniscus has a meniscotibial, or coronary, ligament. It connects the inferior edges of the lateral meniscus to the periphery of the tibial plateau. The lateral meniscus also has a meniscofemoral ligament that extends from the posterior horn of the lateral meniscus to the lateral aspect of the medial femoral condyle. It is given two distinct names, based on its location in relation to the posterior cruciate ligament (PCL). The ligament of Humphrey passes in front of the posterior cruciate ligament. It is less than 1/3 the diameter of the posterior cruciate ligament, but may be confused for the posterior cruciate during arthroscopy. The ligament of Wrisberg passes behind the posterior cruciate ligament, and is about � of the posterior cruciate�s diameter (Figure 104). Its femoral origin often merges with the posterior cruciate ligament. Both ligaments are present in only about 6% of knees. Approximately 70% of people have one or the other of these ligaments, with the majority possessing the more posterior ligament of Wrisberg (Figure 105). MRI is the preferred imaging modality for medial collateral or lateral collateral ligament injuries, as it can detect any associated internal knee derangements, cruciate-collateral ligament injuries, or cartilage deficiencies.

the knee

Figure 104. Rendering of posterior knee, arrow indicates Ligament of Wrisberg; courses obliquely from lateral aspect of medial femoral condyle to posterior horn of lateral meniscus,
remains posterior to PCL.

the knee

Figure 105. Arrow indicates �Wrisberg pseudo-tear�; intermediate signal
intensity line at junction of
Ligament of Wrisberg and normal posterior horn of lateral meniscus; often mistaken for a meniscal tear.

The patellar ligament is the connection between the patella and the tibia, extending from the apex (inferior aspect) of the patella to the tibial tuberosity. Technically, it is connecting two bones, so it is a ligament. However, it is most often referred to as the patellar tendon, because the superficial fibers that cover the front of the patella and extend to the tibia are continuous with the central portion of the common tendon of the quadriceps femoris muscle. The posterior surface of the patellar ligament is separated from the synovial membrane of the knee joint by a large infrapatellar pad of fat. Injuries to the patellar ligament can occur from overuse, such as sports that involve jumping and quick directional changes, as well as running-related sports. This is the ligament that is injured in jumper�s knee (or patellar tendonitis), which begins with inflammation, and can lead to degeneration or rupture of the patellar ligament and the tissue around it (Figure 106). Patients with patellar ligament injuries typically complain of pain in the area below the kneecap, which will increase with walking, running, squatting, etc. They can often be treated in the same manner as other soft tissue injuries- with rest, ice, compression and elevation. The patellar ligament attachment at the tibial tuberosity is the site of Osgood-Schlatter disease, which was discussed previously.

the knee

Figure 106. Patellar tendonitis (jumper�s knee).

Along the sides of the patella and the patellar ligament are the medial and lateral patellar retinacula (Figure 107). They are fibrous tissue stabilizers for the patella that form from the medial and lateral portions of the quad tendons as they pass down to insert on either side of the tibial tuberosity. The lateral retinaculum is the thicker of the two, but both have superficial and deep layers. Within the deep layers are various ligaments (whose names indicate the structures they connect) that help support the patella in its position, relative to the femur below it. The deep layer of the lateral patellar retinaculum is the location where the lateral patellofemoral ligament meets the iliopatellar band, which is a tract of fibers from the iliotibial (IT) band that connects to the patella. The deep layer of the medial patellar retinaculum has three focal capsular thickenings, referred to as the medial patellofemoral, medial patellomeniscal, and medial patellotibial ligaments. The medial patellofemoral ligament is strong enough to influence patellar tracking, and acts as a major medial restraint. Imbalances in the forces that control patellar tracking during flexion and extension of the knee can lead to patellofemoral pain syndrome (runner�s knee), one of the most common causes of knee pain. This can result from overuse, trauma, muscle dysfunction, patellar hypermobility, and poor quadriceps flexibility. Typical symptoms include pain behind or around the patella that is increased with running, and activities that involve knee flexion. MRI is typically not necessary for this diagnosis. Physical therapy has been found to be effective for the treatment of patellofemoral pain syndrome.

the knee

Figure 107. Lateral and medial retinaculum.

Muscles & Tendons Of The Knee

The flexor and extensor muscles of the knee have been discussed previously, as the majority of them are the anterior and posterior muscles of the thigh. We will review the thigh muscles involved in knee movement, and add two muscles of the lower leg that also affect the knee. The quadriceps femoris muscles of the anterior thigh are the main knee extensors (Figure 108). As these muscles contract, the knee joint straightens. The tendons of the vastus medialis, vastus intermedius, vastus lateralis, and rectus femoris join at the superior aspect (base) of the patella to form the patellar tendon. This tendon continues over the patella and attaches it to the tibial tuberosity (since it is connecting bone to bone, it is sometimes called the patellar ligament). The quadriceps, along with the gluteal muscles, are responsible for the thrusting forces necessary for walking, running, and jumping. The quads also help control movement of the patella, as they are attached to it by the quadriceps tendons (Figure 109). The patella increases the force exerted by the quadriceps muscles as the knee is straightened.

the knee

Figure 108. Anterior thigh muscles – knee extensors.

the knee

Figure 109. Quadriceps controlling the patella.

 

 

 

 

 

 

 

 

 

 

 

 

 

The posterior thigh muscles, also known as the hamstrings, are the main knee flexors, with assistance from the sartorius, gracilis, gastrocnemius, and popliteus muscles. The knee bends when the hamstrings contract. The hamstring muscles give the knee joint the strength needed for propulsion in running and jumping. They also help to stabilize the knee by protecting the collateral and cruciate ligaments, especially when the knee twists. The three hamstring muscles have varying attachment sites around the knee joint (Figure 110). The biceps femoris attaches to the head of the fibula and the superolateral aspect of the tibia. The semitendinosus attaches on the anterior aspect of the tibia, medial to the tibial tuberosity, crossing over the medial collateral ligament. The tendon of the semitendinosus muscle is sometimes used for cruciate ligament reconstruction. The semimembranosus attaches at the posteriomedial aspect of the medial tibial condyle. The sartorius muscle is also a knee flexor, although it is an anterior thigh muscle. It inserts on the anterior medical aspect of the tibia. The gracilis muscle of the medial thigh is one of the hip adductors, but also plays a part in knee flexion. Like the semitendinosus tendon, the tendon of the gracilis is sometimes used for cruciate ligament reconstructions. The gracilis attaches to the medial aspect of the proximal tibia.

the knee

Figure 110. Posterior knee
muscles – knee flexors.

Additional flexors of the knee joint include some of the posterior muscles of the lower leg. The large superficial gastrocnemius muscle has a medial and a lateral head, which originate from the medial and lateral femoral condyles, respectively. It runs the length of the posterior lower leg, attaching to the calcaneus by the Achilles tendon. The gastrocnemius gives us the ability to flex our knee while our foot is flexed, as it connects to both joints. It is involved in standing, walking, running, and jumping. The popliteus is a deep posterior lower leg muscle that helps with knee flexion, and also rotates the tibia medially, which aids in knee stability. The popliteus originates from the outer margin of the lateral meniscus of the knee joint. It extends posteriorly and inserts on the medial aspect of the tibia, inferior to the medial tibial epicondyle.

The important tendons of the knee include the quadriceps, patellar, and hamstring tendons, and the iliotibial band (Figure 111). Tendons attach muscles to bones. These major knee tendons have all been discussed with either the bones or the muscles that they attach. The quadriceps tendon was mentioned with the quadriceps muscle as the muscle�s attachment to the patella. The quad tendon continues over the patella, then attaches the apex of the patella to the tibial tuberosity. It is then called the patellar tendon (or ligament). Hamstring tendons were discussed with the hamstring muscles, the posterior muscles that are flexors of the knee. Hamstring tendons are sometimes used for cruciate ligament reconstructions. Tendonitis, which is the inflammation of a tendon, is a common knee injury amongst athletes in a variety of sports. The iliotibial band (or IT tract) functions like a tendon, as it attaches the knee to the tensor fasciae latte muscle. The band is actually a fibrous reinforcement of the fascia lata, or deep tissue of the thigh. It runs from the ilium to the tibia. Proximally, it acts as a hip abductor, while distally it acts as lateral stabilization for the knee, and aids with medial rotation of the tibia. The IT band is in constant use during walking and running, which can lead to irritation at the point where it passes over the lateral femoral epicondyle. A �tight� IT band can cause inflammation and/or irritation at the femoral epicondyle, or at the point of insertion on the lateral tibial condyle. This condition is called IT band friction syndrome. It is common amongst runners, hikers, and cycling enthusiasts.

the knee

Figure 111. Tendons of the knee.

Nerves Of The Knee

The main nerves to the knee that come from the sacral plexus of nerves are the tibial nerve and the common peroneal nerve (Figure 112). Both are branches of the sciatic nerve, and begin posteriorly, slightly above the actual knee joint. Both of these nerves, or their branches, continue through the lower leg and foot, providing sensation and muscle control. The tibial and common peroneal nerves are also both involved in cutaneous innervation, which is the supply of nerves to the skin of the knee. The tibial nerve remains posterior and more medial, branching at the medial ankle to innervate the foot. The common peroneal nerve begins posterolaterally, moving anteriorly near the neck of the fibula. It then branches into the superficial and deep peroneal nerves, which continue their anterior descent to the foot. The tibial and common peroneal nerves are the most commonly injured nerves when a knee is dislocated. Nerves can grow back, but they do so at a rate of approximately � inch per month.

the knee

Figure 112. Sacral plexus nerves of knee.

Nerves from the lumbar plexus that affect the knee include the lateral femoral cutaneous, and the saphenous, which is a branch of the femoral nerve (Figure 113). The saphenous nerve travels more medially and gives off infrapatellar branches around the knee joint. Below the knee, the saphenous nerve sends branches to the skin of the anterior and medial lower leg. The lateral femoral cutaneous nerve sends an anterior branch to the skin of the anterior and lateral thigh, down to the area of the knee. Terminal filaments of this nerve communicate with the infrapatellar branch of the saphenous nerve, forming the peripatellar plexus.

the knee

Figure 113. Lumbar plexus nerves of knee.

Arteries & Veins Of The Knee

The popliteal artery, a branch of the superficial femoral artery, is the main arterial supply to the knee joint. It runs along the posterior aspect of the distal femur, behind the knee joint. At the supracondylar ridge, the popliteal artery gives off the blood supply to the knee, which consists of various genicular arteries (Figure 114). Inferior to the knee joint, the popliteal branches into the anterior and posterior tibial arteries, which supply the lower leg. The popliteal artery is a common site for both atherosclerosis and aneurysms, and is listed as the most common site for peripheral arterial aneurysms. Approximately 50% of these aneurysms are bilateral. Although they rarely rupture, popliteal aneurysms may serve as a focus for abrupt thrombotic occlusion of the involved popliteal artery, which can affect the foot on the same side. A thrombus within an aneurysm can also lead to a distal embolism. The genicular arteries are sources of continued blood flow to the knee and lower limb, in case of an obstructed popliteal artery. The descending genicular, also called the highest or supreme genicular, branches from the femoral artery, just superior to the popliteal branch. It supplies the adductor magnus and hamstring muscles, then joins with the network of genicular arteries around the knee joint. The middle genicular pierces the oblique popliteal ligament, and supplies the ligaments and synovial membrane inside the knee articulation (including the ACL and PCL). The sural artery joins the anastomoses of the genicular arteries, and also supplies muscles of the lower leg, including the large gastrocnemius muscle. The anastomotic pattern around the knee joint is supplied by the popliteal artery posteriorly, the descending genicular artery medially, and the descending branch of the lateral circumflex femoral artery laterally. The genicular arteries involved in the anastomosis are labeled as the medial and lateral superior geniculars, and the medial and lateral inferior geniculars.

the knee

Figure 114. Arteries of knee.

The major deep veins around the knee joint are the popliteal vein, and the anterior and posterior tibial veins (Figure 115). The popliteal vein begins at the junction of the tibial veins in the posterior aspect of the lower leg, just inferior to the knee joint. It ascends posteriorly, continuing as the femoral vein about halfway up the thigh. As deep veins typically follow the arteries, the genicular veins accompany the genicular arteries around the knee joint, then drain into the popliteal vein. The important superficial veins around the knee joint are the small and great saphenous veins. Superficial veins typically do not follow arteries, but rather travel with cutaneous nerves. The small saphenous ascends the lower leg posteriorly, angling from lateral to medial. It merges with the popliteal vein at a position slightly superior to the knee joint. The great saphenous vein, the longest vein in the body, has a medial and anterior course in the lower leg. It moves to a posterior position, but stays medial along the knee joint, moving alongside the medial epicondyle of the femur. The great saphenous then moves anteriorly again through the thigh.

the knee

Figure 115. Veins of knee.

Varicose and �spider� veins are often seen in the leg in the posterior aspect of the knee joint. As mentioned previously, in the femoral vein discussion, veins have valves to ensure the �one-way� uphill flow of blood back to the heart (Figure 116). Communicating vessels, also called perforating veins, exist between the deep and superficial veins to help compensate for valves that may be incompetent, and are allowing blood reflux. If venous walls are weakened or dilated, the cusps of the valves can no longer close properly, and the valves can become incompetent. This leads to an increase in the weight of the column of blood for the veins that are �downstream� from the bad valve. Blood can pool in these veins, causing them to become varicose, where the veins swell, become tortuous, and even bulge through the skin surface. Reticular veins, which are smaller varicose veins that do not bulge through the skin, as well as very small �spider� veins are both typically less severe conditions, but both still involve the backwards flow of blood. Removal of severe varicose veins will actually help blood flow, as the blood will no longer be stagnant in the pooled areas.

the knee

Figure 116. Varicose veins around knee.

Bursae Of The Knee

The synovial knee joint is home to a large number of bursae (Figure 117). These are fluid sacs and synovial pockets that surround and sometimes communicate with the joint cavity. They facilitate friction-free movement between the bones and moving structures (tendon, muscle). Fluid or debris can collect in the bursa, or fluid can extend into the bursa from the adjacent joint in situations such as excessive friction, infection or direct trauma. This type of pathological enlargement of the bursa is referred to as bursitis, which can mimic several peripheral joint and muscle abnormalities. Radiologists must be able to accurately identify bursal pathology, especially amongst the numerous knee bursae (14 reported in some literature). We will identify a few of the more common bursa, beginning with the suprapatellar bursa. This bursa lies between a quadriceps tendon and the femur, superior to the patella (Figure 118). Fluid is commonly found here when patients have a joint effusion. Bursitis of the prepatellar bursa is also known as �housemaid�s knee�. It occurs from repetitive trauma from kneeling, as seen with housemaids, wrestlers, and carpet-layers. This bursa is found between the patella and the skin (Figure 119). Inflammation of the superficial infrapatellar bursa may be called �Clergyman�s knee�, another bursitis that can occur from excessive kneeling. This bursa is located between the distal third of the patellar tendon and the overlying skin (Figure 120).

the knee

Figure 117. Bursae in the knee.

the knee

Figure 118. T2 gradient
displaying suprapatellar
bursa.

the knee

Figure 119. T2
fatsat displaying
prepatellar bursa.

the knee

Figure 120. T2 fatsat
displaying infrapatellar
bursa.

 

The synovial sac of the knee joint sometimes forms a posterior bulge, known as a Baker�s cyst or popliteal cyst (Figure 121). It typically forms between the tendons of the medial head of the gastrocnemius muscle and the semimembranosus muscle, posterior to the medial femoral condyle. Baker�s cysts are not true cysts, as they typically maintain open communication with the synovial sac. However, they can pinch off, and they can rupture. They are usually asymptomatic, but can be indicative of another problem of the knee, such as arthritis or a meniscal tear. Aspiration of the synovial fluid can be performed if the cyst becomes problematic. Treatment is usually necessary if a Baker�s cyst ruptures, as it can cause acute pain behind the knee, and swelling of the calf muscles. A ruptured cyst can also mimic a DVT or thrombophlebitis. Ultrasound and MRI can both be used for confirmation of a Baker�s cyst (Figure 122).

the knee

Figure 121. Lateral view of Baker�s cyst.

the knee

Figure 122. Sagittal image of Baker�s cyst on MRI.

Scan Setups

The following are HMSA suggestions for knee imaging. Knee protocols should be designed to yield diagnostic images of the menisci, bones, articular cartilage, and all ligamentous structures of the knee. While many radiologists may require additional imaging of the ACL, protocols that are designed for optimal imaging of the cartilage and menisci should also produce adequate images of the ACL. Always check with your radiologist for his/her imaging preferences.

Axial Scans

When positioning axial slices for the knee, sagittal and coronal images can be used to insure inclusion of all pertinent anatomy. The slices should extend superiorly to include the entire patella, and inferiorly to include the tibial tuberosity and patellar tendon insertion. A presat can be placed over the unaffected lower extremity to reduce the possibility of wrap-around artifact, as seen in the coronal image in Figure 139.

the knee

Figure 139. Axial slice setup using sagittal and coronal images.

Coronal Scans

Coronal slices of the knee should include the anatomy from the posterior femoral condyles to the anterior portion of the patella. Visualize a line connecting the lateral and medial condyles of the femur. Typically, the coronal slices are angled so that they are parallel to that line, as seen in the axial image in Figure 140.

the knee

Figure 140. Coronal slice setup using axial and sagittal images.

Sagittal Scans

Sagittal slices should include the anatomy from the medial condyle to the lateral condyle. The slice group may be angled per your radiologist�s preference, but should remain perpendicular to the coronal slices. Typically, the slice group is angled so that it is parallel to the medial border of the femoral condyle, as seen in the axial image in Figure 141.

the knee

Figure 141. Sagittal slice setup using axial and coronal images.

In addition to routine oblique sagittal images, some radiologists prefer an additional sagittal scan of the ACL with thin slices and high spatial resolution. Axial and coronal images can be used for slice setup. Referenced literature recommends that the angle of the slice group should not exceed 10� from a line drawn perpendicular to the bicondylar line (line that connects the posterior femoral condyles), as seen in Figure 142.

the knee

Figure 142. Sagittal ACL slice setup using axial and coronal images.

 

blank
References:

Kapit, Wynn, and Lawrence M. Elson. The Anatomy Coloring Book. New York: HarperCollins, 1993.

Hip Anatomy, Function, and Common Problems. (Last updated 28July2010). Retrieved from healthpages.org/anatomy-function/hip-structure-function-common-problems/

Cluett, J. M.D. (Updated 22May2012). Labral Tear of the Hip Joint. Retrieved from orthopedics.about.com/od/hipinjuries/qt/labrum.htm

Hughes, M. D.C. (15July2010). Diseases of the Femur Bone. Retrieved from www.livestrong.com/article/175599-diseases-of-the-femur-bone/

A Patient�s Guide to Perthes Disease of the hip. (n.d.). Retrieved from www.orthopediatrics.com/docs/Guides/perthes.html

Hip Injuries and Disorders. (Last reviewed 10February2012). Retrieved from nlm.nih.gov/medlineplus/hipinjuriesanddisorders.html

Ligament of head of femur. (Updated 20December2011). Retrieved from en.wikipedia.org/wiki/Ligament_of_head_of_femur

Ewing�s sarcoma. (Last modified 06January2012). Retrieved from en.wikipedia.org/wiki/Ewing%27s_sarcoma

Hip Anatomy. (n.d.). Retrieved from www.activemotionphysio.ca/Injuries-Conditions/Hip

Iliotibial Band Friction Syndrome. (n.d.). Retrieved from www.physiotherapy-treatment.com/iliotibial-band-friction-syndrome.html

Snapping hip syndrome. (Last modified 09November2011). Retrieved from en.wikipedia.org/wiki/Snapping_hip_syndrome

Sekul, E. (Updated 03February2012). Meralgia Paresthetica. Retrieved from emedicine.medscape.com/article/1141848-overview

Yeomans, S. D.C. (Updated 07July2010). Sciatic Nerve and Sciatica. Retrieved from www.spine-health.com/conditions/sciatica/sciatic-nerve-and-sciatica

Mayo Clinic staff. (26July2011). Meralgia paresthetica. Retrieved from www.mayoclinic.com/health/meralgia-paresthetica/DS00914

Deep Vein Thrombosis (DVT)-Blood Clots in the Legs. (n.d.). Retrieved from catalog/nucleusinc.com/displaymonograph.php?MID=148

Petersilge, C. M.D. (03May2000). Chronic Adult Hip Pain: MR Arthrography of the Hip. Retrieved from radiographics.rsna.org/content/20/suppl_1/S43.full

Acetabular branch of medial circumflex femoral artery. (Last modified 17November2011). Retrieved from en.wikipedia.org/wiki/Acetabular_branch_of_medial_circumflex_femoral_artery

Cluett, J. M.D. (Updated 26March2011). Hip Bursitis. Retrieved from orthopedics.about.com/cs/hipsurgery/a/hipbursitis.htm

Steinbach, L. M.D., Palmer, W. M.D., Schweitzer, M. M.D. (10June2002). Special Focus Session MR Arthrography. Retrieved from radiographics.rsna.org/content/22/5/1223.full

Schueler, S. M.D., Beckett, J.M.D., Gettings, S.M.D. (Last updated 05August2010). Ischial Bursitis/Overview. Retrieved from www.freemd.com/ischial-bursitis/overview.htm

Hwang, B., Fredericson, M., Chung, C., Beaulieu, C., Gold, G. (29October2004). MRI Findings of Femoral Diaphyseal Stress Injuries in Athletes. Retrieved from www.ajronline.org/content/185/1/166.full.pdf

The Femur (Thigh Bone). (n.d.). Retrieved from education.yahoo.com/reference/gray/subjects/subject/59

Norman, W. PhD, DSc. (n.d.). Joints of the Lower Limb. Retrieved from home.comcast.net/~wnor/lljoints.htm

Femur. (Last modified 24September2012). Retrieved from en.wikipedia.org/wiki/Femur

Wheeless, C. III, M.D. (Last updated 25April2012). Ligaments of Humphrey and Wrisberg. Retrieved from wheelessonline.com/ortho/ligaments_of_humphrey_and_wrisberg

Muscle Strains in the Thigh. (Last reviewed August2007). Retrieved from orthoinfo.aaos.org/topic.cfm?topic=A00366

Shiel, W. Jr., M.D. (Last reviewed 23July2012). Hamstring Injuries. Retrieved from www.medicinenet.com/hamstring_injury/article.htm

Hamstring Muscle Injuries. (Last reviewed July 2009). Retrieved from orthoinfo.aaos.org/topic.cfm?topic=a00408

Knee. (Last modified 19September2012). Retrieved from en.wikipedia.org/wiki/Knee

DeBerardino, T. M.D. (Updated 30March2012). Quadriceps Injury. Retrieved from emedicine.medscape.com/article/91473-overview

Kan, J.H. (n.d.). Osteochondral Abnormalities: Pitfalls, Injuries, and Osteochondritis Dissecans. Retrieved from www.arrs.org/shopARRS/products/s11p_sample.pdf

Nerves of the Lower Limb. (Last updated 30March2006). Retrieved from download.videohelp.com/vitualis/med/lowrnn.htm

The Adductor Canal. (Last updated 30March2006). Retrieved from download.videohelp.com/vitualis/med/addcanal.htm

Nabili, S. M.D. (n.d.). Varicose Veins & Spider Veins. Retrieved from www.medicinenet.com/varicose_veins/article.htm

Basic Venous Anatomy. (n.d.). Retrieved from vascular-web.com/asp/samples/sample104.asp

Femoral nerve. (Last modified 23September2012). Retrieved from en.wikipedia.org/wiki/Femoral_nerve

Peron, S. RDCS. (Last modified 16October2010). Anatomy � Lower Extremity Veins. Retrieved from www.vascularultrasound.net/vascular-anatomy/veins/lower-extremity-veins

Medical Multimedia Group, L.L.C. (n.d.). Knee Anatomy. Retrieved from www.eorthopod.com/content/knee-anatomy

Knee Joint Anatomy, Function and Problems. (Last updated 06July2010). Retrieved from healthpages.org/anatomy-function/knee-joint-structure-function-problems/

Coronary ligament of the knee. (Last modified 09May2010). Retrieved from en.wikipedia.org/wiki/Coronary_ligament_of_the_knee

Walker, B. (n.d.). Patellar Tendonitis Treatment � Jumper�s Knee. Retrieved from www.thestretchinghandbook.com/archives/patellar-tendonitis.php

Osgood-Schlatter disease. (Last reviewed 12November2010). Retrieved from www.ncbi.nlm.nih.gov/pubmedhealth/PMH0002238/

Grelsamer, R. M.D. (n.d.). The Anatomy of the Patella and the Extensor Mechanism. Retrieved from kneehippain.com/patient_pain_anatomy.php

Oblique popliteal ligament. (Last modified 24March2012). Retrieved from en.wikipedia.org/wiki/Oblique_popliteal_ligament

Shiel, W. Jr., M.D. (Last reviewed 27July2012). Chondromalacia Patella (Patellofemoral Syndrome). Retrieved from www.medicinenet.com/patellofemoral_syndrome/article.htm

Knee. (Last modified 19September2012). Retrieved from en.wikipedia.org/wiki/Knee

Mosher, T. M.D. (Last updated 11April2011). MRI of Knee Extensor Mechanism Injuries Overview of the Knee Extensor Mechanism. Retrieved from emedicine.medscape.com/article/401001-overview

Carroll, J. M.D. (December 2007). Oblique Menisco-meniscal Ligament. Retrieved from radsource.us/clinic/0712

DeBerardino, T. M.D. (Last updated 30March2012). Medial Collateral Knee Ligament Injury. Retrieved from emedicine.medscape.com/article/89890-overview#a0106

Farr, G. (Last updated 31December2007). Joints and Ligaments of the Lower Limb. Retrieved from becomehealthynow.com/article/bodyskeleton/951/

Knee anatomy overview. (02March2008). Retrieved from www.kneeguru.co.uk/KNEEnotes/node/741

Dixit, S. M.D., Difiori, J. M.D., Burton, M. M.D., Mines, B. M.D. (15January2007). Management of Patellofemoral Pain Syndrome. Retrieved from www.aafp.org/afp/2007/0115/p194.html

Knee Muscles. (Last updated 05September2012). Retrieved from www.knee-pain-explained.com/kneemuscles.html

Popliteus muscle. (Last updated 20February2012). Retrieved from en.wikipedia.org/wiki/Popliteus_muscle

Kneedoc. (10February2011). Nerves. Retrieved from thekneedoc.co.uk/neurovascular/nerves

Wheeless, C. III, M.D. (Last updated 15December2011). Popliteal Artery. Retrieved from wheelessonline.com/ortho/popliteal_artery

The Popliteal Artery. (n.d.) Retrieved from education.yahoo.com/reference/gray/subjects/subject/159

Knee bursae. (Last updated 09May2012). Retrieved from en.wikipedia.org/wiki/Bursae_of_the_knee_joint

Hirji, Z., Hunjun, J., Choudur, H. (02May2011). Imaging of the Bursae. Retrieved from www.ncbi.nlm.nih.gov/pmc/articles/PMC3177464/

Kimaya Wellness Limited. (n.d.). Organ>Popliteal Artery. Retrieved from kimayahealthcare.com/OrganDetail.aspx?OrganID=103&AboutID=1

Total Vein Care. (Last updated 24February2012). Varicose Vein Anatomy and Function for Patients. Retrieved from www.veincare.com/education/

Tibia. (Last updated 01April2012). Retrieved from en.wikipedia.org/wiki/Tibia

Norkus,S., Floyd, R. (Published 2001). The Anatomy and Mechanisms of Syndesmotic Ankle Sprains. Retrieved from www.ncbi.nlm.nih.gov/pmc/articles/PMC155405/

Soleus muscle. (Last updated 10April2012). Retrieved from en.wikipedia.org/wiki/Soleus_muscle

Achilles Tendinitis. (Last reviewed June2010). Retrieved from orthoinfo.aaos.org/topic.cfm?topic=A00147

Wheeless, C. III,M.D. (Last updated 11April2012). Sural Nerve. Retrieved from wheelessonline.com/ortho/sural_nerve

Medical Multimedia Group, L.L.C. (Last updated 26July2006). Ankle Syndesmosis Injuries. Retrieved from www.orthogate.org/patient-education/ankle/ankle-syndesmosis-injuries.html

Cluett, J. M.D. (Last updated 16September2008). Exertional Compartment Syndrome. Retrieved from orthopedics.about.com/od/overuseinjuries/a/compartment.htm

Leg Veins (Thigh, Lower Leg) Anatomy, Pictures and Names. (Last updated 21November2010). Retrieved from www.healthype.com/leg-veins-thigh-lower-leg-anatomy-pictures-and-names.html

Cluett, J.M.D. (Last updated 6October2009). Stress Fracture. Retrieved from orthopedics.about.com/cs/otherfractures/a/stressfracture.htm

Ostlere, S. (1December2004). Imaging the ankle and foot. Retrieved from imaging.birjournals.org/content/15/4/242.full

Inverarity, L. D.O. (Last updated 23January2008). Ligaments of the Ankle Joint. Retrieved from physicaltherapy.about.com/od/humananatomy/p/ankleligaments.htm

Golano, P., Vega, J., DeLeeuw, P., Malagelada, F.,Manzanares, M., Gotzens, V., van Dijk, C. (Published online 23March2010). Anatomy of the ankle ligaments:a pictorial essay. Retrieved from www.ncbi.nlm.nih.gov/pmc/articles/PMC2855022/

Numkarunarunrote, N., Malik, A., Aguiar, R.,Trudell, D., Resnick, D. (11October2006). Retinacula of the Foot and Ankle: MRI with Anatomic Correlation in Cadavers. Retrieved from www.ajronline.org/content/188/4/W348.full

Medical Multimedia Group, L.L.C. (n.d.). A Patient�s Guide to Ankle Anatomy. Retrieved from www.eorthopod.com/content/ankle-anatomy

The Anterior Tibial Artery. (n.d.). Retrieved from education.yahoo.com/reference/gray/subjects/subject/160

Foot and Ankle Anatomy. (Last updated 28July2011). Retrieved from northcoastfootcare.com/pages/Foot-and-Ankle-Anatomy.html

Donnelly, L., Betts, J., Fricke, B. (1July2009). Skimboarder�s Toe: Findings on High-Field MRI. Retrieved from www.ajronline.org/content/184/5/1481.full

Foot. (Last updated 28August2012). Retrieved from en.wikipedia.org/wiki/Foot

Wiley, C. (n.d.). Major Ligaments in the Foot. Retrieved from www.ehow.com/list_6601926_major-ligaments-foot.html

Turf Toe: Symptoms, Causes, and Treatments. (Last reviewed 9August2012). Retrieved from www.webmd.com/fitness-exercise/turf-toe-symptoms-causes-and-treatments

Cluett, J. M.D. (Last updated 02April2012). Turf Toe. Retrieved from orthopedics.about.com/od/toeproblems/p/turftoe.htm

Neurology and the Feet. (n.d.) Retrieved from footdoc.ca/www.FootDoc.ca/Website%20Nerves%20Of%20The%20Feet.htm

The Veins of the Lower Extremity, Abdomen, and Pelvis. (n.d.). Retrieved from education.yahoo.com/reference/gray/subjects/subject/173

Corley, G., Broderick, B., Nestor, S., Breen, P., Grace, P., Quondamatteo, F., O�Laighin, G. (n.d.). The Anatomy and Physiology of the Venous Foot Pump. Retrieved from www.eee.nuigalway.ie/documents/go_anatomy_of_the_plantar_venous_plexus_manuscript.pdf

Morton�s neuroma. (Last modified 8August2012). Retrieved from en.wikipedia.org/wiki/Morton%27s_metatarsalgia

References For Anatomy Pics:

Figures 1, 5, 6, 24- www.orthopediatrics.com/docs/Guides/perthes.html

Figures 2, 3, 11, 12, 14, 15, 16, 18, 23, 25- www.activemotionphysio.ca/Injuries-Conditions/Hip/Hip-Anatomy/a~299/article.html

Figure 4- hipkneeclinic.com/images/uploaded/hipanatomy_xray.jpg

Figures 7, 8, 9- hipfai.com/

Figure 10- en.wikipedia.org/wiki/File:Ewing%27s_sarcoma_MRI_nci-vol-1832-300.jpg

Figure 13- www.chiropractic-help.com/Patello-Femoral-Pain-Syndrome.html

Figure 17- www.thestretchinghandbook.com/archives/ezine_images/adductor.jpg

Figure 19- media.summitmedicalgroup.com/media/db/relayhealth-images/hipanat.jpg

Figures 20-22- www.ajronline.org/content/182/1/137.full.pdf+html

Figure 43, 44- radiographics.rsna.org/content/20/suppl_1/S43.full

Figure 45- www.exploringnature.org/db/detail.php?dbID=24&detID=2768

Figures 46-48- www.ajronline.org/content/185/1/166.full.pdf

Figure 49- arrs.org/shopARRS/products/s11p_sample.pdf

Figure 50- www.thestretchinghandbook.com/archives/medial-collateral-ligament.php

Figures 51, 52- www.radsource.us/clinic/0712

Figures 53, 54- www.osteo-path.co.uk/BodyMap/Thighs.html

Figure 55- www.ncbi.nlm.nih.gov/pmc/articles/PMC1963576/

Figure 56- legacy.owensboro.kctcs.edu/gcaplan/anat/Notes/API%20Notes%20M%20%20Peripheral%20Nerves.htm

Figure 57- www.keywordpictures.com/keyword/lateral%20cutaneous%20nerve%20of%20thigh/

Figure 58- home.comcast.net/~wnor/postthigh.htm

Figure 59- becomehealthynow.com/glossary/CONG437.htm

Figure 60- fitsweb.uchc.edu/student/selectives/Luzietti/Vascular_pvd.htm

Figure 61- www.fashion-res.com/peripheral-vascular-disease-with-stenting-in-the/

Figure 62- www.wpclipart.com/medical/anatomy/blood/femoral_artery_and_branches_in_leg.png.html

Figure 63- www.globalteleradiologyservices.com/Deep_Vein_Thrombosis_Overview.htm

Figure 64- www.vascularultrasound.net/vascular-anatomy/veins/lower-extremity-veins

Figure 82- www.jeffersonhospital.org/diseases-conditions/knee-ligament-injury.aspx?disease=658f267f-75ab-4bde-8781-f2730fafa958

Figure 83- javierjuan.ifunnyblog.com/anatomybackofknee/

Figure 84- www.kneeandshouldersurgery.com/knee-disorders/tibial-osteotomy.html

Figure 85- www.disease-picture.com/chondromalacia-patella-physical-therapy/

Figure 86- www.eorthopod.com/content/bipartite-patella

Figure 87- www.orthogate.org/patient-education/knee/articular-cartilage-problems-of-the-knee.html

Figure 88- www.webmd.com/pain-management/knee-pain/menisci-of-the-knee-joint

Figure 89- sumerdoc.blogspot.com/2008_07_01_archive.html

Figure 90- www.concordortho.com/patient-education/topic-detail-popup.aspx?topicID=55befba2d440dc8e25b85747107b5be0

Figure 91- trialx.com/curebyte/2011/08/16/pictures-for-chondromalacia-patella/

Figure 92- radiopaedia.org/images/1059

Figure 93- radiologycases.blogspot.com/2011/01/osgood-schlatter-disease.html

Figure 94- www.physioquestions.com/2010/09/07/knee-injury-acl-part-i/

Figure 95- www.jeffersonhospital.org/diseases-conditions/knee-ligament-injury.aspx?disease=4e3fcaf5-0145-43ea-820f-a175e586e3c8

Figures 96, 97- radiology.rsna.org/content/213/1/213.full

Figures 98-101- appliedradiology.com/Issues/2008/12/Articles/Imaging-the-knee–Ligaments.aspx

Figure 102- radiopaedia.org/images/408156

Figure 103- aftabphysio.blogspot.com/2010/08/joints-of-lower-limb.html

Figures 104, 105- www.radsource.us/clinic/0310

Figure 106- nwrunninglab.com/patellar-tendonitis.html

Figure 107- www.aafp.org/afp/2007/0115/p194.html

Figure 108- www.reboundsportspt.com/blog/tag/knee-pain

Figure 109- www.norwellphysicaltherapy.com/Injuries-Conditions/Knee/Knee-Issues/Quadriceps-Tendonitis-of-the-Knee/a~1803/article.html

Figure 110- kneeguru.co.uk/KNEEnotes/node/479

Figure 111- www.magicalrobot.org/BeingHuman/2010/03/fascia-bones-and-muscles

Figure 112- home.comcast.net/~wnor/postthigh.htm

Figures 113, 115, 157-159- ipodiatry.blogspot.com/2010/02/anatomy-of-foot-and-ankle_26.html

Figure 114- medchrome.com/basic-science/anatomy/the-knee-joint/

Figure 116- www.sharecare.com/question/what-are-varicose-veins

Figure 117- mendmyknee.com/knee-and-patella-injuries/anatomy-of-the-knee.php

Figures 118-120- www.ncbi.nlm.nih.gov/pmc/articles/PMC3177464/

Figure 121- www.riversideonline.com/health_reference/Disease-Conditions/DS00448.cfm

Figure 122- arthritis.ygoy.com/2011/01/01/what-is-an-arthritis-knee-cyst/

Figure 143- usi.edu/science/biology/mkhopper/hopper/BIOL2401/LABUNIT2/LabEx11week6/tibiaFibulaAnswer.htm

Figure 144- web.donga.ac.kr/ksyoo/department/education/grossanatomy/doc/html/fibula1.html

Figure 145- becomehealthynow.com/popups/ligaments_tib_fib_bh.htm

Figure 146- www.parkwayphysiotherapy.ca/article.php?aid=121

Figure 147- aidmyankle.com/high-ankle-sprains.php

Figure 148- legsonfire.wordpress.com/what-is-compartment-syndrome/

Figures 149, 152- www.stepbystepfootcare.ca/anatomy.html

Figures 150, 151- www.gla.ac.uk/ibls/US/fab/tutorial/anatomy/jiet.html

Figure 153- www.athletictapeinfo.com/?s=tennis+leg

Figure 154- radsource.us/clinic/0608

Figure 155- www.eorthopod.com/content/achilles-tendon-problems

Figure 156- achillesblog.com/assumptiondenied/not-a-rupture/

Figure 181- www.orthopaedicclinic.com.sg/ankle/a-patients-guide-to-ankle-anatomy/

Figure 182- www.activemotionphysio.ca/article.php?aid=47

Figure 183- www.ajronline.org/content/193/3/687.full

Figures 184, 186- www.eorthopod.com/content/ankle-anatomy

Figure 185- www.crossfitsouthbay.com/physical-therapy/learn-yourself-a-quick-anatomy-reference/ankle/

Figures 187, 227- www.activemotionphysio.ca/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html

Figure 188- inmotiontherapy.com/article.php?aid=124

Figures 189, 190- home.comcast.net/~wnor/ankle.htm

Figure 191- skillbuilders.patientsites.com/Injuries-Conditions/Ankle/Ankle-Anatomy/a~47/article.html

Figure 192- metrosportsmed.patientsites.com/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html

Figure 193- musc.edu/intrad/AtlasofVascularAnatomy/images/CHAP22FIG30.jpg

Figure 194- musc.edu/intrad/AtlasofVascularAnatomy/images/CHAP22FIG31B.jpg

Figure 195- veinclinics.com/physicians/appearance-of-vein-disease/

Figure 196- mdigradiology.com/services/interventional-services/varicose-veins.php

Figure 216- kidport.com/RefLib/Science/HumanBody/SkeletalSystem/Foot.htm

Figure 217- www.joint-pain-expert.net/foot-anatomy.html

Figure 218- www.thetoedoctor.com/turf-toe-symptoms-and-treatment/

Figures 219, 220- radsource.us/clinic/0303

Figure 221- www.ajronline.org/content/184/5/1481.full

Figure 222- www.answers.com/topic/arches

Figure 223- www.mayoclinic.com/health/medical/IM00939

Figure 224- radsource.us/clinic/0904

Figure 225- www.ortho-worldwide.com/anfobi.html

Figure 226- www.coringroup.com/lars_ligaments/patientscaregivers/your_anatomy/foot_and_ankle_anatomy/

Figure 228- www.stepbystepfootcare.ca/anatomy.html

Figure 229- iupucbio2.iupui.edu/anatomy/images/Chapt11/FG11_18aL.jpg

Figure 230- www.ajronline.org/content/184/5/1481.full.pdf

Figure 231- metrosportsmed.patientsites.com/Injuries-Conditions/Foot/Foot-Anatomy/a~251/article.html

Figure 232- www.painfreefeet.com/nerve-entrapments-of-the-leg-and-foot.html

Figures 233, 234- emedicine.medscape.com/article/401417-overview

Figure 235- web.squ.edu.om/med-Lib/MED_CD/E_CDs/anesthesia/site/content/v03/030676r00.HTM

Figure 236- www.nysora.com/peripheral_nerve_blocks/classic_block_tecniques/3035-ankle_block.html

Figure 237- ultrasoundvillage.net/imagelibrary/cases/?id=122&media=464&testyourself=0

Figure 238- www.joint-pain-expert.net/foot-anatomy.html

Figure 239- jap.physiology.org/content/109/4/1045.full

Figure 240- microsurgeon.org/secondtoe

Figure 241- elu.sgul.ac.uk/rehash/guest/scorm/406/package/content/common_iliac_veins.htm

Close Accordion